Orthodontic Treatment Time: Factors That Affect Duration

Orthodontic treatment duration varies substantially among patients and cannot be accurately predicted from appliance type alone.

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Comprehensive treatment with fixed appliances commonly extends over approximately 18–24 months, although individual treatment times may be considerably shorter or longer depending on the initial malocclusion, treatment objectives, biomechanics, and patient-related factors.

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A systematic review of prospective studies reported a mean duration of approximately 19.9 months for comprehensive fixed-appliance treatment, whereas another systematic review found a mean duration of approximately 24.9 months across a broader range of clinical trials. These differences illustrate the heterogeneity of orthodontic treatment protocols and patient populations.

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🔹 How Long Does Orthodontic Treatment Take?
For comprehensive treatment, a practical clinical estimate is often around 18–24 months, but this should be considered a general range rather than a fixed treatment time.
Treatment duration depends on the objectives of therapy. Limited alignment may require substantially less time, whereas comprehensive correction involving extractions, significant sagittal discrepancies, vertical problems, impacted teeth, or complex space management may require considerably longer treatment.

🔹 Factors That Influence Orthodontic Treatment Duration

Factor	Potential Effect on Treatment Time
Initial malocclusion severity	Greater discrepancy and tooth irregularity may require longer correction
Dental crowding	Increased irregularity can prolong alignment
Tooth extractions	Space closure and finishing may extend treatment
Impacted teeth	Surgical exposure and orthodontic traction can substantially increase duration
Skeletal discrepancies	Complex orthopedic or surgical approaches may require additional phases
Treatment mechanics	Anchorage requirements and biomechanics influence the sequence and rate of tooth movement
Patient compliance	Missed appointments, poor elastic wear, and appliance breakage can delay progress
Oral hygiene	Poor hygiene may require treatment interruptions or modifications
Treatment complications	Root resorption, periodontal problems, or unexpected tooth movement can alter the treatment plan
Clinician and treatment factors	Planning, biomechanics, monitoring, and clinical efficiency can affect treatment progression

1. Initial Malocclusion and Treatment Complexity
The severity and complexity of the initial malocclusion are among the most important determinants of treatment duration. Greater crowding, larger anteroposterior discrepancies, complex rotations, vertical problems, and extensive space requirements generally increase the number of movements required to achieve the treatment objectives.
Evidence from a systematic review of tooth alignment demonstrated that baseline irregularity significantly affects alignment time. In individual patient data analyses, each additional millimeter of initial irregularity was associated with approximately 17.5 additional days required for whole-arch mandibular alignment.

2. Extractions and Space Closure
Extraction-based orthodontic treatment may require additional time because the treatment must incorporate controlled space closure, root positioning, anchorage management, and final occlusal detailing.
Systematic-review evidence has identified extraction treatment as a factor associated with longer treatment duration, although the magnitude of the effect varies according to the malocclusion and mechanics used. Four-premolar extraction treatment has also been associated with increased treatment duration in clinical-trial evidence.

3. Impacted Teeth
Impacted teeth, particularly maxillary canines, can substantially prolong orthodontic treatment. Management may require surgical exposure, orthodontic traction, space creation, and subsequent alignment.
The presence of impacted maxillary canines has consistently been identified as a factor associated with increased treatment duration.

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4. Patient Compliance
Patient cooperation can directly influence treatment efficiency. Missed appointments, inadequate elastic wear, poor adherence to removable appliances, and repeated bracket or appliance failures can interrupt planned biomechanics.
Evidence concerning removable appliances demonstrates that actual wear time is frequently lower than prescribed, highlighting the importance of adherence when treatment depends on patient-controlled appliance use.

5. Age and Biological Response
Age is often considered a determinant of orthodontic treatment time; however, current evidence requires a more nuanced interpretation.
A systematic review comparing adolescents and adults found no significant difference in overall comprehensive fixed-appliance treatment duration between the groups. However, adult patients with palatally displaced canines required longer canine alignment in the available evidence. The certainty of these findings was limited by substantial heterogeneity and the predominantly non-randomized nature of the available studies.
Therefore, chronological age alone should not be used to predict treatment duration without considering the specific clinical situation.

6. Orthodontic Appliance and Technique
The assumption that one orthodontic appliance or bracket prescription consistently produces substantially shorter treatment is not strongly supported by current evidence.
Systematic reviews have found limited evidence for clinically important differences in treatment duration between different fixed-appliance prescriptions and techniques. More recent evidence also suggests that bracket slot size may have little or no clinically significant effect on overall treatment duration, although the certainty of evidence is low.
Similarly, evidence comparing clear aligners and fixed appliances in mild-to-moderate crowding has not demonstrated a consistent significant difference in treatment duration.

7. Treatment Interruptions and Clinical Complications
Unexpected events can extend treatment beyond the original estimate. These include broken appliances, missed appointments, inadequate oral hygiene, periodontal complications, delayed eruption, unfavorable tooth movement, and the need to modify treatment mechanics.
Consequently, an initial treatment estimate should be regarded as a clinical projection rather than a guaranteed completion date.

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💬 Discussion
Orthodontic treatment duration is multifactorial. Although a comprehensive fixed-appliance treatment period of approximately 18–24 months is frequently encountered clinically, published systematic reviews demonstrate variability in mean duration depending on study design, patient characteristics, malocclusion severity, and treatment protocol.
The available evidence indicates that case complexity, initial irregularity, extraction requirements, impacted teeth, patient cooperation, and treatment interruptions can have meaningful effects on treatment time. In contrast, claims that a particular bracket system or prescription inherently produces substantially shorter treatment are not consistently supported by high-quality evidence.
Treatment planning should therefore prioritize the biological and mechanical requirements of the individual malocclusion rather than using appliance selection as the principal method for reducing treatment duration.

✍️ Conclusion
Orthodontic treatment time is determined by the interaction of biological, clinical, mechanical, and patient-related factors. Comprehensive treatment frequently requires approximately 18–24 months, but complex cases may require substantially longer periods.
Accurate treatment-time estimation should consider malocclusion severity, crowding, extractions, impacted teeth, treatment mechanics, compliance, and potential complications. Current evidence does not support assuming that a particular appliance system will automatically produce a shorter treatment period.

🎯 Clinical Recommendations
▪️ Establish a case-specific treatment-time range rather than promising a fixed completion date.
▪️ Identify factors likely to prolong treatment before appliance placement, particularly severe crowding, extractions, impacted teeth, and complex skeletal discrepancies.
▪️ Incorporate anticipated anchorage and space-closure requirements into the treatment estimate.
▪️ Monitor compliance, appliance integrity, and appointment attendance because preventable interruptions can accumulate over the course of treatment.
▪️ Reassess the projected completion date periodically when treatment response differs from the initial biomechanical plan.

📚 References

✔ Abbing, A., Koretsi, V., Eliades, T., & Papageorgiou, S. N. (2020). Duration of orthodontic treatment with fixed appliances in adolescents and adults: A systematic review with meta-analysis. Progress in Orthodontics, 21, 37. https://doi.org/10.1186/s40510-020-00334-4
✔ Papageorgiou, S. N., Höchli, D., & Eliades, T. (2017). Outcomes of comprehensive fixed appliance orthodontic treatment: A systematic review with meta-analysis and methodological overview. Korean Journal of Orthodontics, 47(6), 401–413. https://doi.org/10.4041/kjod.2017.47.6.401
✔ Papageorgiou, S. N., Koletsi, D., Iliadi, A., Peltomäki, T., & Eliades, T. (2017). Treatment effects of various prescriptions and techniques for fixed orthodontic appliances: A systematic review. European Journal of Orthodontics, 39(6), 599–610. https://doi.org/10.1093/ejo/cjx020
✔ Tsichlaki, A., Chin, S. Y., Pandis, N., & Fleming, P. S. (2016). How long does treatment with fixed orthodontic appliances last? A systematic review. American Journal of Orthodontics and Dentofacial Orthopedics, 149(3), 308–318. https://doi.org/10.1016/j.ajodo.2015.09.020
✔ Wazwaz, F., Seehra, J., Carpenter, G. H., Ireland, A. J., Papageorgiou, S. N., & Cobourne, M. T. (2022). Duration of tooth alignment with fixed appliances: A systematic review and meta-analysis. American Journal of Orthodontics and Dentofacial Orthopedics, 161(1), 20–36. https://doi.org/10.1016/j.ajodo.2021.06.016

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Medications That Interfere With Orthodontics: Clinical Guide

Orthodontic tooth movement (OTM) is a biologically regulated process that depends on periodontal ligament responses, inflammatory mediators, osteoclast activity, and coordinated alveolar bone remodeling.

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Because several medications influence these pathways, systemic pharmacotherapy can potentially modify the rate of orthodontic tooth movement, anchorage, treatment duration, or management of orthodontic pain.

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The clinical relevance varies substantially between drug classes. Current evidence identifies nonsteroidal anti-inflammatory drugs (NSAIDs) and antiresorptive medications, particularly bisphosphonates, as the most important groups to recognize during orthodontic treatment. However, much of the available evidence remains heterogeneous, with considerable reliance on animal and experimental studies.

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🔹 How Medications Can Affect Orthodontic Tooth Movement
Orthodontic movement requires bone resorption on the pressure side and bone formation on the tension side. Medications that modify prostaglandin production, osteoclast differentiation, or systemic bone turnover may therefore alter the biological response to orthodontic forces.
The principal clinically relevant medications are summarized below.

Medication Class	Examples	Potential Effect on OTM	Clinical Relevance
NSAIDs	Ibuprofen, aspirin, diclofenac, ketorolac, meloxicam	May reduce movement by inhibiting prostaglandin-mediated bone remodeling	Most relevant with repeated or prolonged use
Bisphosphonates	Alendronate, risedronate, zoledronic acid	Reduced osteoclast activity and potentially slower OTM	Important in long-term therapy; requires individualized assessment
Other Antiresorptives	Denosumab and related agents	Potential modification of bone turnover and OTM	Clinical evidence is less established than for bisphosphonates
Corticosteroids	Prednisone, dexamethasone	May modify bone remodeling; effects are variable	Greater consideration with chronic systemic therapy
Hormonal and Bone-Metabolism Agents	Thyroid hormone, vitamin D, parathyroid hormone analogues	Potential alteration of bone turnover and OTM	Evidence is mainly experimental or heterogeneous
Other Systemic Medications	Statins, metformin, propranolol and others	Variable effects reported in experimental studies	Insufficient evidence for routine clinical modification of treatment

1. Nonsteroidal Anti-Inflammatory Drugs
NSAIDs are among the most clinically relevant medications because prostaglandins participate in the inflammatory and bone-remodeling processes associated with orthodontic movement.
Experimental and clinical evidence indicates that some NSAIDs can reduce the rate of OTM. Aspirin, diclofenac, ketorolac, and nimesulide have demonstrated inhibitory effects, whereas findings for ibuprofen, meloxicam, and celecoxib are more inconsistent and appear to depend on dose, duration, and route of administration.
Importantly, the evidence does not support avoiding NSAIDs categorically. They remain effective for orthodontic pain, and short-term use does not necessarily produce a clinically meaningful reduction in tooth movement. The 2026 systematic review of acetaminophen versus ibuprofen also confirms their role in orthodontic pain control, while the broader literature continues to show uncertainty regarding their effect on OTM.
Acetaminophen (paracetamol) differs pharmacologically from traditional NSAIDs and has generally demonstrated less interference with orthodontic tooth movement. It has therefore been extensively investigated as an alternative for orthodontic pain management.

2. Bisphosphonates and Antiresorptive Medications
Bisphosphonates inhibit osteoclast-mediated bone resorption and represent the medication group with the most consistently documented inhibitory effect on orthodontic tooth movement.
Alendronate, risedronate, and other bisphosphonates can reduce bone turnover and potentially slow orthodontic tooth movement. A 2026 umbrella review found that all included systematic reviews reported reduced OTM following bisphosphonate administration, although most of the underlying evidence was derived from animal studies. Human evidence remains limited and does not allow firm conclusions regarding the magnitude of the clinical effect.
This issue is particularly relevant in adults receiving long-term antiresorptive therapy for osteoporosis, Paget disease, or malignancy-associated bone disease. The medication history should be documented before initiating orthodontic treatment.

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3. Corticosteroids
Systemic corticosteroids can influence bone remodeling and osteoblast–osteoclast activity. However, their effect on orthodontic tooth movement is less predictable than that of NSAIDs or bisphosphonates.
Short-term and long-term corticosteroid exposure should therefore not be interpreted identically. Chronic systemic therapy may have greater implications for bone metabolism and periodontal health, but the available evidence is insufficient to establish a uniform effect on orthodontic movement.

4. Hormonal and Bone-Metabolism Medications
Medications and agents that modify systemic bone turnover—including thyroid hormones, vitamin D, parathyroid hormone analogues, and sex-hormone-related therapies—have been investigated as potential modifiers of OTM.
Some experimental studies suggest acceleration or inhibition of tooth movement depending on the agent and biological context. Nevertheless, the clinical evidence is heterogeneous, and most findings cannot currently justify changing orthodontic mechanics solely because a patient uses one of these medications.

5. Other Common Medications
Several commonly prescribed drugs—including statins, metformin, propranolol, calcium compounds, losartan, and some gastrointestinal medications—have demonstrated effects on orthodontic tooth movement in animal studies. However, the quality of evidence is generally low, and findings cannot be directly extrapolated to routine human orthodontic care.
Therefore, these medications should be considered part of the patient's pharmacological history rather than automatically classified as contraindications to orthodontic treatment.

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💬 Discussion
The relationship between medications and orthodontic tooth movement is biologically plausible but clinically complex. The strongest evidence concerns medications that directly modify inflammatory signaling or osteoclast-mediated bone resorption. NSAIDs may interfere with prostaglandin-dependent remodeling, while bisphosphonates have a more pronounced antiresorptive mechanism.
Nevertheless, the overall certainty of evidence remains limited. A 2026 systematic review included 64 studies and concluded that much of the available evidence was derived from preclinical models, with overall certainty ranging from low to very low. Consequently, laboratory findings should not automatically be translated into changes in clinical orthodontic protocols.
The practical implication is not that patients taking these medications should routinely discontinue therapy or be excluded from orthodontic treatment. Rather, medication history should be integrated into orthodontic diagnosis, treatment planning, and monitoring. Any modification or discontinuation of a systemic medication should be determined by the prescribing physician or appropriate medical specialist.

✍️ Conclusion
Several medications can potentially interfere with orthodontic tooth movement, primarily through effects on prostaglandin signaling, osteoclast activity, or systemic bone remodeling. NSAIDs and bisphosphonates have the clearest evidence of potential inhibitory effects, whereas the clinical significance of corticosteroids and other systemic medications remains less certain.
A comprehensive medication history is therefore an important component of orthodontic assessment, particularly in adults receiving chronic pharmacological therapy. Current evidence supports individualized monitoring rather than routine alteration or discontinuation of medically indicated medications.

🎯 Clinical Recommendations
▪️ Obtain a complete medication history before initiating orthodontic treatment and update it during treatment.
▪️ Identify patients receiving long-term bisphosphonate or other antiresorptive therapy and assess the indication, duration, route, and medical context.
▪️ For orthodontic pain, consider the shortest effective duration of analgesic therapy, taking the patient's general medical status into account.
▪️ Do not discontinue or alter a medically necessary medication solely to accelerate orthodontic tooth movement; coordinate such decisions with the prescribing physician.
▪️ In patients receiving medications that may alter bone remodeling, monitor tooth movement response and treatment progress rather than assuming a predictable treatment delay.
▪️ Interpret evidence from animal studies cautiously because the clinical magnitude of pharmacological effects on human OTM remains uncertain.

📚 References

▪️ Giannini, L., Macrì, F., Inchingolo, A. M., Inchingolo, F., Dipalma, G., & Maspero, C. (2026). Influence of pharmacological agents on orthodontic tooth movement: A systematic review. Bioengineering, 13(2), 224. https://doi.org/10.3390/bioengineering13020224
▪️ Amin, S., Cremona, M., & Abela, S. (2026). Effect of bisphosphonates on orthodontic tooth movement: An umbrella review. BMC Oral Health. https://doi.org/10.1186/s12903-026-08984-2
▪️ Colceriu-Șimon, I.-M., Feștilă, D., Emoke, H., Pancsur, A., Șimon, M. Ș., Olteanu, C. D., Păstrav, M., Bunta, O., & Ghergie, M. (2025). The effects of non-steroidal anti-inflammatory drugs used for orthodontic pain management on tooth movement: A comprehensive review of the literature. Journal of Clinical Medicine, 14(9), 2920. https://doi.org/10.3390/jcm14092920
▪️ Neves, N. M., Rodrigues, Á. O. L. J., Bordin, G. M., Occhi-Alexandre, I. G. P., Orsi, J. S. R., Gabardo, M. C. L., & Topolski, F. (2026). Effectiveness of acetaminophen in comparison with ibuprofen for pain control in orthodontic patients: A systematic review and meta-analysis. Korean Journal of Orthodontics, 56(3), 187–199. https://doi.org/10.4041/kjod25.273
▪️ Makrygiannakis, M. A., Kaklamanos, E. G., & Athanasiou, A. E. (2018). Does common prescription medication affect the rate of orthodontic tooth movement? A systematic review. European Journal of Orthodontics, 40(6), 649–659. https://doi.org/10.1093/ejo/cjy001
▪️ Rakhshan, V. (2017). The influence of non-steroidal anti-inflammatory drugs and paracetamol used for pain control of orthodontic tooth movement: A systematic review. Dental Press Journal of Orthodontics, 22(5), 64–72.

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MBT vs Damon Brackets: Clinical Differences Explained

Choosing between MBT brackets and Damon brackets involves more than comparing conventional and self-ligating bracket designs. These systems differ in bracket mechanics, ligation, archwire engagement, and treatment philosophy, while clinical outcomes are also strongly influenced by diagnosis, treatment objectives, wire sequence, anchorage, and operator technique.

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MBT brackets are conventional preadjusted edgewise brackets that use elastomeric or metal ligatures to secure the archwire. The MBT prescription is widely used in fixed orthodontic treatment and incorporates specific bracket angulations and torque values designed to support straight-wire mechanics.

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Damon brackets, in contrast, are self-ligating brackets that use a built-in clip or sliding mechanism to secure the archwire. Damon systems have been associated with low-friction mechanics and simplified archwire engagement, although systematic reviews have not demonstrated consistent clinical superiority over conventional brackets across major treatment outcomes.
Understanding the distinction between these systems is therefore important when selecting an appliance for a specific orthodontic treatment plan.

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🔹 What Are MBT Brackets?
The MBT bracket system is a preadjusted edgewise appliance derived from modifications of the Andrews straight-wire concept. Its prescription incorporates specific values of torque, angulation, and in-out positioning for individual teeth.
Unlike self-ligating systems, conventional MBT brackets require an external ligation method. Elastomeric modules are commonly used, although stainless-steel ligatures may also be selected depending on the clinical objective.
MBT brackets can be used for a broad range of malocclusions and treatment approaches, including extraction and non-extraction treatment.

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🔹 What Are Damon Brackets?
Damon brackets are a family of self-ligating orthodontic brackets developed around a passive or low-friction bracket mechanism. The defining feature is the integrated door or clip that holds the archwire without requiring an elastomeric ligature.
The Damon approach has traditionally emphasized reduced friction, lighter orthodontic forces, and the use of specific archwire sequences. However, the clinical effects of self-ligation should not be interpreted solely from laboratory friction measurements because tooth movement in vivo involves biological, mechanical, and periodontal factors.
Clinical trials comparing Damon and conventional brackets have not consistently demonstrated shorter overall treatment times or superior occlusal outcomes.

🔹 MBT vs Damon Brackets: Key Differences

Feature	MBT Brackets	Damon Brackets
Bracket type	Conventional preadjusted	Self-ligating
Archwire retention	Elastomeric or metal ligature	Integrated clip or door
Ligation	Required	Not required
Friction	Influenced by ligature and wire-bracket interaction	Designed for low-friction engagement
Prescription	MBT prescription	Damon-specific prescription
Archwire engagement	Controlled with ligatures	Controlled by bracket mechanism
Chairside ligation	Required	Reduced
Alignment	Effective with conventional mechanics	Effective with self-ligating mechanics
Treatment time	Primarily influenced by case complexity and mechanics	No consistent overall reduction demonstrated
Pain/discomfort	Variable	Variable
Anchorage control	Depends on biomechanics and appliance configuration	Depends on biomechanics and appliance configuration
Finishing	Requires conventional finishing mechanics	Requires conventional finishing mechanics
Clinical selection	Based on prescription and treatment objectives	Based on system design and treatment objectives

🔹 Ligation and Friction
One of the most obvious differences between MBT vs Damon brackets is how the archwire is secured.
In conventional MBT brackets, elastomeric ligatures can increase resistance to sliding between the bracket and archwire. Damon brackets eliminate the need for these external ligatures by incorporating a mechanical locking mechanism.
This distinction can influence the mechanics of sliding and the time required for archwire engagement. However, lower laboratory friction does not automatically translate into faster orthodontic treatment.
A systematic review of self-ligating brackets found evidence supporting reduced chair time in some circumstances, but did not identify consistent advantages in overall treatment time or final occlusal characteristics.

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🔹 Alignment Efficiency
Orthodontic alignment is frequently cited when comparing conventional and self-ligating brackets.
Clinical evidence is mixed. A randomized clinical trial directly comparing Damon3 and MBT brackets found greater improvement in upper anterior irregularity during a four-month alignment period with Damon3 brackets. However, the difference in lower anterior alignment over the complete observation period was not statistically significant. Pain levels were also not significantly different between the groups.
Other randomized clinical research has produced different results. In one trial comparing Damon3 with conventional brackets, no significant difference in the initial rate of alignment was identified.
Therefore, Damon brackets should not automatically be considered faster for alignment in every clinical situation.

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🔹 Treatment Time
A major reason clinicians and patients may consider self-ligating brackets is the possibility of reducing total treatment time.
However, evidence does not consistently support this claim.
A randomized clinical trial involving extraction patients found no significant effect of Damon3 versus conventional brackets on overall treatment duration, number of visits, or overall occlusal improvement.
Systematic reviews have similarly reported insufficient evidence for a clinically important reduction in total treatment duration with self-ligating systems.

Treatment duration remains strongly influenced by factors such as:
▪️ Initial malocclusion severity
▪️ Extraction requirements
▪️ Anchorage demands
▪️ Space closure
▪️ Patient compliance
▪️ Missed appointments
▪️ Appliance breakages
▪️ Treatment mechanics
▪️ Finishing requirements

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🔹 Pain and Patient Comfort
Patient discomfort is another commonly discussed difference between MBT and Damon brackets.
The available evidence does not establish a consistent pain advantage for self-ligating brackets. A systematic review found no significant difference in pain between self-ligating and conventional appliances at several assessment intervals.
Similarly, the randomized Damon3-versus-MBT study found no statistically significant difference in pain experience between the two bracket groups.
Pain is therefore better considered a patient-specific response influenced by the magnitude and type of orthodontic force, archwire changes, tooth movement, and individual sensitivity.

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🔹 Arch Expansion and Transverse Changes
Damon treatment is sometimes associated with the concept of achieving greater arch development through low-friction mechanics.
However, clinical research does not support assuming that self-ligating brackets automatically produce greater transverse changes.
A multicenter randomized controlled trial comparing passive self-ligating, active self-ligating, and conventional brackets found no significant differences in maxillary transverse dimensional changes attributable to bracket type.
This distinction is clinically important. Changes in arch width should be planned according to the patient's anatomy, periodontal limits, tooth position, skeletal relationships, and treatment objectives rather than attributed solely to the bracket system.

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🔹 Biomechanical Considerations
The bracket system is only one component of orthodontic biomechanics.
With MBT brackets, elastomeric or steel ligation allows the clinician to control how the archwire is engaged. Different ligation strategies can therefore be used according to the treatment phase.
Damon brackets simplify archwire engagement through the self-ligating mechanism. Their design may reduce the need for repeated ligature placement and can be useful in situations where efficient archwire engagement is desirable.
Nevertheless, force systems are determined by the interaction between bracket prescription, archwire dimensions, material properties, bracket positioning, ligation, anchorage, and the biological response to force.
Consequently, changing from MBT to Damon does not replace the need for sound biomechanical planning.

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🔹 Clinical Comparison: MBT vs Damon
From a clinical perspective, neither system should be selected solely because of claims about reduced friction or faster treatment.

MBT may be particularly useful when:
▪️ Conventional ligation is preferred.
▪️ Detailed control of archwire engagement is required.
▪️ Specific ligation strategies are part of the treatment mechanics.
▪️ The clinician routinely uses the MBT prescription.
▪️ Conventional finishing and torque-control strategies are preferred.

Damon may be particularly useful when: ▪️ A self-ligating mechanism is preferred.
▪️ Reduced ligation time is clinically valuable.
▪️ The clinician incorporates Damon-specific mechanics into treatment.
▪️ Simplified archwire engagement is desirable.
▪️ A low-friction bracket design is preferred as part of the overall appliance strategy.
These considerations do not establish one system as universally superior. Appliance selection should be based on the malocclusion, treatment objectives, biomechanics, and clinician experience.

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🔹 Does Damon Treat Faster Than MBT?
Current evidence does not support a general statement that Damon brackets treat orthodontic cases faster than MBT brackets.
Some individual studies have reported differences during particular phases, such as initial alignment. However, randomized trials and systematic reviews have generally failed to demonstrate a consistent reduction in overall treatment duration with self-ligating brackets.
This distinction between phase-specific efficiency and overall treatment efficiency is important when interpreting claims about self-ligating appliances.

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🔹 MBT vs Damon: Which Factors Matter Most?
When comparing these systems, clinicians should consider:

1. Diagnosis: Skeletal and dental relationships determine the fundamental treatment strategy.
2. Treatment objectives: Alignment, space closure, torque control, anchorage, and finishing may require different mechanics.
3. Bracket prescription: Torque and angulation values affect tooth positioning.
4. Archwire sequence: Wire size, material, and progression influence force delivery.
5. Ligation: Conventional ligation allows different methods of archwire engagement.
6. Anchorage: Bracket selection does not eliminate the need for appropriate anchorage control.
7. Patient factors: Compliance and appointment attendance can substantially influence treatment duration.
8. Clinician technique: Bracket positioning and biomechanical execution remain critical.

🔹 Evidence-Based Clinical Perspective
The available evidence suggests that self-ligating brackets can reduce some chairside ligation procedures, but the evidence for clinically meaningful advantages in overall treatment time, pain, occlusal outcomes, or arch development is inconsistent.
A randomized clinical trial specifically comparing Damon3 and MBT brackets demonstrated that differences can occur during the alignment phase, but these findings should not be generalized to every orthodontic case.
Therefore, the most appropriate comparison is not simply "Which bracket is better?" but rather "Which bracket system fits the treatment objectives and biomechanics of this case?"

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🎯 Clinical Recommendations
▪️ Select the bracket system according to the diagnosis and treatment objectives, not marketing claims.
▪️ Do not assume that lower friction automatically means shorter overall treatment.
▪️ Consider the advantages of conventional ligation when specific archwire engagement or force control is required.
▪️ Consider self-ligating brackets when their mechanical design and reduced ligation requirements fit the clinician's workflow.
▪️ Evaluate arch expansion and tooth movement within the patient's biological and periodontal limits.
▪️ Use evidence from randomized trials and systematic reviews when evaluating claims about treatment efficiency.
▪️ Remember that bracket positioning, archwire selection, anchorage, and biomechanics can be more important than the bracket mechanism alone.

✍️ Conclusion
The comparison of MBT vs Damon brackets highlights two different approaches to fixed orthodontic appliance design. MBT brackets use conventional ligation and a preadjusted prescription, while Damon brackets incorporate a self-ligating mechanism designed to simplify archwire engagement and reduce the need for external ligatures.
Although some studies have reported differences in specific treatment phases, current evidence does not establish a consistent overall clinical advantage of Damon brackets over conventional systems in treatment duration, pain, or final occlusal outcomes.
The decision should therefore be based on the malocclusion, treatment objectives, biomechanics, archwire sequence, anchorage requirements, and clinician preference.
For orthodontists comparing bracket prescriptions and treatment philosophies, the next useful comparison is [Roth vs MBT brackets], which examines how these two conventional preadjusted prescriptions differ in torque, angulation, bracket design, and clinical application. Understanding Roth vs MBT provides an important foundation before comparing conventional systems such as MBT with self-ligating systems such as Damon.

📚 References

▪️ Jahanbin, A., Hasanzadeh, N., Khaki, S., & Shafaee, H. (2019). Comparison of self-ligating Damon3 and conventional MBT brackets regarding alignment efficiency and pain experience: A randomized clinical trial. Journal of Dental Research, Dental Clinics, Dental Prospects, 13(4), 281–288.
▪️ Chen, S. S. H., Greenlee, G. M., Kim, J. E., Smith, C. L., & Huang, G. J. (2010). Systematic review of self-ligating brackets. American Journal of Orthodontics and Dentofacial Orthopedics, 137(6), 726.e1–726.e18.
▪️ Papageorgiou, S. N., et al. (2017). Therapeutic efficacy of self-ligating brackets: A systematic review. Orthodontics & Craniofacial Research.
▪️ Fleming, P. S., Johal, A., & Pandis, N. (2013). Self-ligating brackets in orthodontics: A systematic review. Angle Orthodontist.
▪️ DiBiase, A. T., Nasr, I. H., Scott, P., & Cobourne, M. T. (2011). Duration of treatment and occlusal outcome using Damon3 self-ligated and conventional orthodontic bracket systems in extraction patients: A prospective randomized clinical trial. American Journal of Orthodontics and Dentofacial Orthopedics, 139(2), e111–e116.
▪️ Papageorgiou, S. N., et al. (2021). Are self-ligating brackets more efficient than conventional brackets? A meta-analysis of randomized controlled and split-mouth trials. International Orthodontics.
▪️ Fleming, P. S., et al. (2012). Systematic review on self-ligating vs. conventional brackets: Initial pain, number of visits, and treatment time.

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Acute vs Chronic Odontogenic Infections in Children

Odontogenic infections in children most commonly originate from dental caries, pulpal necrosis, trauma, or periodontal disease. Their clinical behavior ranges from a localized process with minimal systemic involvement to rapidly progressive facial cellulitis or deep-space infection.

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For the pediatric dentist, distinguishing an acute odontogenic infection from a chronic odontogenic infection is clinically important because the presentation, urgency, risk of dissemination, and treatment strategy may differ.

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Importantly, the apparent absence of pain does not necessarily indicate resolution: chronic infections may drain through a sinus tract and remain associated with a necrotic tooth.
Current pediatric guidance emphasizes that treatment should primarily address the source of infection, while systemic antibiotics are reserved for appropriately selected cases rather than used routinely for localized dental disease.

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✅ Acute vs Chronic Odontogenic Infections
An acute infection generally develops over a relatively short period and may produce pain, swelling, tenderness, fever, malaise, or rapidly progressive facial inflammation. A chronic infection may persist for weeks or longer and can present with intermittent symptoms, a draining sinus tract, localized swelling, or radiographic evidence of periapical or furcation pathology.

Feature	Acute Odontogenic Infection	Chronic Odontogenic Infection
Onset	Rapid	Gradual or prolonged
Pain	Frequently present	May be absent or intermittent
Swelling	Often prominent	Usually localized or intermittent
Drainage	May be absent initially	Sinus tract or spontaneous drainage may occur
Systemic signs	Possible fever, malaise, or lymphadenopathy	Usually absent unless acute exacerbation occurs
Typical dental source	Acute pulpal or periapical infection	Persistent necrotic pulp or chronic periapical/furcation infection
Primary concern	Rapid local or systemic spread	Persistent infection and possible acute exacerbation
Main treatment principle	Prompt source control ± antibiotics when indicated	Definitive elimination of the dental source

The distinction should not be based solely on symptom duration. Clinical examination, dental history, radiographic findings, and assessment of systemic involvement are required to establish the diagnosis.

✅ Clinical Assessment
The evaluation should establish both the odontogenic source and the severity of infection.

Important findings include:
▪️ Pain: spontaneous versus provoked, intensity, duration, and progression.
▪️ Soft-tissue swelling: localized versus diffuse.
▪️ Sinus tract: particularly important in chronic infection.
▪️ Tooth mobility: excessive mobility unrelated to normal exfoliation or trauma may indicate pulpal necrosis or infection.
▪️ Percussion and palpation: useful when clinically obtainable.
▪️ Lymphadenopathy: may indicate regional inflammatory involvement.
▪️ Fever and malaise: suggest systemic involvement.
▪️ Trismus, dysphagia, or respiratory difficulty: potential indicators of extension into deeper spaces and require urgent escalation.
▪️ Radiographic findings: furcation/periapical radiolucency, pathologic root resorption, or other evidence of infection.
In primary and immature permanent teeth, conventional thermal and electric pulp tests can be unreliable; diagnosis therefore requires integration of history, clinical findings, and radiographic assessment.

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✅ Acute Odontogenic Infections: Clinical Approach
Acute infections may range from a localized abscess to facial cellulitis.
When infection is localized and there are no systemic signs or evidence of spreading infection, definitive dental treatment is the central intervention. Depending on the tooth and prognosis, this may include appropriate pulp therapy or extraction.
AAPD guidance identifies acute facial swelling of dental origin as a condition requiring particular attention because infection can progress beyond the local tissues.

When Antibiotics May Be Appropriate
Systemic antibiotics should generally be considered when there is evidence of:

▪️ Facial cellulitis or progressive diffuse swelling
▪️ Fever or other systemic manifestations
▪️ Regional lymphadenopathy associated with spreading infection
▪️ Significant extraoral involvement
▪️ Deep-space infection or risk of rapid progression
▪️ Situations in which definitive source control cannot immediately be achieved and systemic involvement is present
Antibiotics are adjunctive therapy, not a substitute for treatment of the infected tooth. Contemporary pediatric recommendations emphasize antimicrobial stewardship and avoidance of routine antibiotics for localized dental conditions without systemic involvement.

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✅ Chronic Odontogenic Infections in Children
Chronic infection may be clinically subtle. A sinus tract, recurrent localized swelling, or an asymptomatic radiolucency may represent an ongoing odontogenic infection.
A draining sinus tract should not be interpreted as resolution. Instead, it frequently represents a pathway through which purulent material is being decompressed while the underlying dental source persists. Chronic odontogenic sinus tracts in children have been associated with prolonged infection from caries or trauma and may occasionally present extraorally.
For a primary tooth with irreversible pulpitis or necrotic pulp, current AAPD guidance supports nonvital pulp treatment when the tooth is restorable and clinically appropriate. Pulpectomy and lesion sterilization/tissue repair are among the available approaches, with treatment selection influenced by root resorption, restorability, prognosis, and the child's developmental needs.
When the infectious process cannot be predictably controlled, the tooth is unrestorable, or there is extensive pathological root resorption, extraction should be considered.

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✅ Antibiotics: What the Pediatric Dentist Should Remember
One of the most important distinctions is between infection requiring dental treatment and infection requiring systemic antimicrobial therapy.
A systematic review specifically evaluating antibiotics for odontogenic abscesses in children with primary teeth found insufficient clinical evidence to establish benefit in localized abscesses without systemic involvement.
The current AAPD recommendations similarly emphasize that antibiotics should be prescribed selectively and that antimicrobial stewardship is essential because unnecessary exposure contributes to adverse effects and antimicrobial resistance.

Therefore:
Localized dental infection → prioritize definitive dental treatment.
Spreading/systemic infection → definitive dental treatment + appropriately selected systemic antibiotic therapy when indicated.

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✅ Red Flags Requiring Urgent Escalation
The following findings should prompt urgent referral or hospital-based assessment, depending on severity:

▪️ Rapidly progressive facial or cervical swelling
▪️ Difficulty breathing
▪️ Dysphagia or inability to swallow secretions
▪️ Significant trismus
▪️ Floor-of-mouth elevation
▪️ Toxic appearance or marked systemic illness
▪️ Orbital involvement
▪️ Suspected deep neck-space infection
▪️ Failure to respond to appropriate initial management
Pediatric odontogenic facial cellulitis can rarely become life-threatening. Recent multidisciplinary literature emphasizes source control, appropriate antimicrobial therapy, and hospital management for severe or complicated cases.
Imaging is particularly useful when there is concern for deep-space involvement, abscess formation, or complications that cannot be adequately evaluated clinically.

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💬 Discussion
The distinction between acute and chronic odontogenic infection is clinically useful, but these categories should not be regarded as completely separate disease entities. A chronic infection may undergo an acute exacerbation, producing sudden pain and swelling in a previously asymptomatic tooth.
The principal clinical issue is therefore not simply the duration of symptoms but the relationship between pulpal status, local tissue involvement, systemic response, and potential anatomical spread.
The evidence consistently supports source control as the foundation of treatment. Antibiotics cannot reliably eradicate a persistent odontogenic focus when the infected tooth remains untreated. This principle is particularly relevant in children, in whom inappropriate antibiotic prescribing can expose patients to adverse effects without addressing the underlying dental pathology.
Another important consideration is that the evidence base specifically addressing antibiotic treatment of odontogenic abscesses in children remains limited. Consequently, antibiotic decisions should be based on the clinical severity of infection, systemic involvement, patient-specific factors, and contemporary pediatric antimicrobial guidance rather than on the mere presence of pus or a dental abscess.

✍️ Conclusion
Acute and chronic odontogenic infections in children require different levels of clinical vigilance, but both demand identification and elimination of the dental source.
Acute infections require careful assessment for rapid progression, facial cellulitis, and systemic involvement, whereas chronic infections may be deceptively asymptomatic and frequently present through sinus drainage or radiographic changes.
For both presentations, definitive dental treatment remains the cornerstone of management. Systemic antibiotics should be reserved for cases in which clinical findings indicate systemic involvement, spreading infection, or other appropriate indications. Early recognition of red flags and timely escalation are essential to prevent severe complications.

🎯 Clinical Recommendations
1. Determine the pulpal and periapical diagnosis before prescribing antibiotics.
2. Treat the dental source as soon as clinically feasible rather than relying on antimicrobial therapy alone.
3. Consider systemic antibiotics primarily when infection is spreading or systemic signs are present.
4. Do not interpret a draining sinus tract as resolution of the underlying infection.
5. Assess restorability, root resorption, tooth value, and developmental considerations when selecting pulp therapy versus extraction.
6. Escalate urgently when airway compromise, dysphagia, significant trismus, rapidly progressive swelling, or deep-space involvement is suspected.
7. Document clinical findings, diagnosis, treatment, antibiotic indication when applicable, and follow-up.
These recommendations are consistent with current AAPD guidance emphasizing individualized diagnosis, definitive source control, and antimicrobial stewardship.

📚 References

✔ American Academy of Pediatric Dentistry. (2026). Use of antibiotic therapy for pediatric dental patients. In The Reference Manual of Pediatric Dentistry. American Academy of Pediatric Dentistry.
✔ American Academy of Pediatric Dentistry. (2026). Pulp therapy for primary and immature permanent teeth: Indications and objectives. In The Reference Manual of Pediatric Dentistry. American Academy of Pediatric Dentistry.
✔ American Academy of Pediatric Dentistry. (2020). Use of non-vital pulp therapies in primary teeth. Pediatric Dentistry, 42(5), 337–349.
✔ Clarke, R. (2023). Pediatric odontogenic and paranasal sinus infections. Neuroimaging Clinics of North America, 33(4), 673–684. https://doi.org/10.1016/j.nic.2023.05.014
✔ Coll, J. A., Dhar, V., Chen, C.-Y., et al. (2024). Use of vital pulp therapies in primary teeth 2024. Pediatric Dentistry, 46(1), 13–26.
✔ Welti, R., et al. (2025). Pediatric odontogenic facial cellulitis: A comprehensive review for multidisciplinary management. Journal of the Pediatric Infectious Diseases Society. https://doi.org/10.1093/jpids/piaf108
✔ Wong, A., et al. (2021). Are systemic antibiotics indicated in children presenting with an odontogenic abscess in the primary dentition? A systematic review of the literature. International Journal of Paediatric Dentistry.

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Roth Technique in Orthodontics: Step-by-Step Treatment

The Roth technique in orthodontics is a pre-adjusted edgewise approach developed from the Straight-Wire Appliance concept, incorporating specific bracket prescriptions for tip, torque, and in-out positioning.

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Its treatment philosophy places particular emphasis on achieving appropriate tooth position, functional occlusion, and a stable finishing result.

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Roth's publications also emphasized the relationship between orthodontic tooth positioning and functional occlusion, including mandibular position, occlusal contacts, anterior guidance, and elimination of undesirable interferences.
Importantly, contemporary evidence does not support considering the Roth prescription as inherently superior to other pre-adjusted systems. Clinical outcomes depend on diagnosis, bracket positioning, biomechanics, anchorage control, wire selection, finishing, and clinician execution. Comparative studies have found no significant differences in several treatment outcomes between Roth and other prescriptions such as MBT.

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🔹 What Is the Roth Technique?
The Roth technique uses pre-adjusted brackets designed to incorporate three-dimensional information into the appliance:

▪️ Tip: controls mesiodistal crown angulation.
▪️ Torque: influences labiolingual or buccolingual inclination.
▪️ In-out: controls the buccolingual position of the tooth.
▪️ Bracket positioning: determines how accurately the prescription is transferred to the dentition.
The prescription is intended to reduce the amount of compensatory wire bending required during treatment. However, bracket positioning and individual tooth morphology can produce clinically relevant deviations from the intended prescription, making finishing adjustments frequently necessary.

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🔹 Step-by-Step Roth Treatment Protocol

1. Diagnosis and Treatment Planning
Treatment begins with a comprehensive diagnosis rather than with bracket selection.

The clinician should evaluate:
▪️ Skeletal and dental relationships
▪️ Facial proportions and soft-tissue profile
▪️ Transverse and vertical dimensions
▪️ Overjet and overbite
▪️ Crowding and spacing
▪️ Incisor inclination and position
▪️ Molar and canine relationships
▪️ Periodontal status
▪️ Temporomandibular and functional findings
▪️ Anchorage requirements
The treatment plan should establish whether treatment will be non-extraction, extraction-based, orthodontic-orthopedic, or combined with other approaches.
The Roth philosophy traditionally gives considerable importance to functional occlusion and the relationship between tooth position and mandibular function.

2. Bracket Selection and Accurate Placement
After establishing the treatment objectives, the appropriate Roth prescription brackets are selected.
Accurate bracket placement is critical because the built-in prescription can only express its intended effect when the bracket is positioned appropriately.

Bracket positioning should consider:
▪️ Clinical crown morphology
▪️ Vertical height
▪️ Mesiodistal position
▪️ Tooth rotation
▪️ Individual tooth anatomy
▪️ Planned finishing position
When significant anatomical variation exists, the clinician may need to compensate through bracket repositioning, archwire adjustments, or auxiliary mechanics.

3. Initial Alignment and Leveling
The first active stage focuses on alignment and leveling.
Flexible nickel-titanium archwires are commonly used to progressively align teeth while controlling excessive force levels. The exact wire sequence should be individualized according to the initial malocclusion rather than treated as a universal Roth protocol.

Typical objectives include:
1. Correct rotations.
2. Reduce initial irregularity.
3. Establish coordinated arch forms.
4. Begin leveling the curve of Spee when appropriate.
5. Prepare the dentition for rectangular working wires.
Evidence concerning specific archwire sequences indicates that no single sequence has been established as universally superior.

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4. Working-Archwire Stage
Once adequate alignment has been achieved, progressively larger rectangular archwires can be introduced.

This stage is used for greater control of:
▪️ Torque
▪️ Tip
▪️ Arch coordination
▪️ Vertical relationships
▪️ Root positioning
Rectangular stainless-steel or other appropriate working wires can provide increased control for space closure and finishing mechanics.
The transition should be based on the degree of alignment, periodontal considerations, bracket engagement, and the mechanical requirements of the case.

5. Space Management and Anchorage Control
In extraction cases, space closure becomes a major component of treatment.

Depending on the diagnosis and anchorage requirements, space may be managed through:
▪️ Canine retraction
▪️ En-masse anterior retraction
▪️ Sequential space closure
▪️ Sliding mechanics
▪️ Elastomeric chains
▪️ NiTi closing coils
▪️ Temporary anchorage devices when indicated
Anchorage control should be planned before space closure begins, particularly when anterior retraction or molar position is critical.
Current evidence indicates that different space-closure auxiliaries can produce broadly similar rates of closure, emphasizing the importance of biomechanical control rather than reliance on a particular auxiliary.

6. Occlusal Correction
After alignment and space management, treatment progresses toward correction of the sagittal, vertical, and transverse relationships.

Depending on the diagnosis, mechanics may include:
▪️ Class II or Class III elastics
▪️ Vertical elastics
▪️ Interarch coordination
▪️ Archwire adjustments
▪️ Auxiliary mechanics
▪️ Controlled molar movement
The objective is not simply to achieve an acceptable static bite but to establish appropriate interarch relationships and functional contacts.

7. Finishing and Detailing
Finishing is particularly important in a pre-adjusted appliance because the bracket prescription represents an average prescription rather than an individualized guarantee of final tooth position.

The clinician should evaluate:
▪️ Incisor inclination
▪️ Root parallelism
▪️ Marginal ridge relationships
▪️ Tooth rotations
▪️ Overjet
▪️ Overbite
▪️ Midlines
▪️ Canine and molar relationships
▪️ Arch coordination
▪️ Occlusal contacts
▪️ Functional excursions
Small bends, bracket repositioning, elastics, or finishing auxiliaries may be necessary.
The original Roth philosophy emphasized functional occlusal relationships, including appropriate contacts and the absence of undesirable excursive interferences.

8. Retention
After active treatment, retention should be planned according to the patient's initial malocclusion, treatment mechanics, periodontal conditions, and individual relapse risk.

Possible approaches include:
▪️ Fixed bonded retainers
▪️ Removable retainers
▪️ Combination retention
Retention is not simply the final step of appliance therapy; it is an essential component of maintaining the achieved tooth positions.

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🔹 Roth Technique: Clinical Sequence

Treatment Stage	Primary Objective	Main Clinical Consideration
Diagnosis	Define skeletal, dental, and functional problems	Individualized treatment plan
Bracket placement	Transfer the prescription accurately	Position and morphology
Alignment	Correct irregularity and rotations	Controlled force application
Working wires	Control torque, tip, and arch coordination	Biomechanical requirements
Space closure	Manage extraction or existing spaces	Anchorage control
Finishing	Optimize tooth position and occlusion	Individual detailing
Retention	Maintain treatment results	Relapse risk and patient factors

💬 Discussion
The Roth technique in orthodontics provides a structured framework for fixed-appliance treatment by incorporating pre-adjusted bracket information into the appliance and emphasizing the relationship between tooth position and occlusion.
However, the prescription itself should not be interpreted as an independent determinant of treatment quality. A systematic review of randomized clinical trials concluded that evidence supporting robust clinical superiority of one fixed-appliance prescription or technique over another remains limited.
Similarly, comparative research involving Roth and MBT prescriptions has not demonstrated consistent clinically meaningful differences in final anterior inclination or subjective aesthetic outcomes.
Consequently, the clinical value of the Roth approach lies in the integration of diagnosis, bracket positioning, biomechanics, occlusal objectives, and individualized finishing, rather than in the prescription alone.

✍️ Conclusion
The Roth technique remains an important pre-adjusted edgewise approach in contemporary orthodontic education and clinical practice. Its stepwise application involves diagnosis, accurate bracket placement, alignment and leveling, working-wire control, space management, occlusal correction, finishing, and retention.
Its successful application requires recognizing that the built-in bracket prescription is only a starting point. Individual tooth anatomy, treatment objectives, biomechanics, and finishing adjustments ultimately determine how closely the intended prescription is expressed clinically.

🎯 Clinical Recommendations
▪️ Use the Roth prescription as a treatment framework, not as a substitute for individualized diagnosis.
▪️ Prioritize accurate bracket positioning, because bracket placement directly affects the expression of tip, torque, and in-out values.
▪️ Establish anchorage requirements before initiating space closure.
▪️ Assess tooth position and occlusion continuously rather than assuming the prescription will automatically produce the desired final result.
▪️ Reserve finishing adjustments for documented discrepancies in torque, angulation, rotation, marginal ridge alignment, and occlusal contacts.
▪️ Interpret claims of superiority among bracket prescriptions cautiously because current evidence does not establish a consistent clinical advantage for one pre-adjusted prescription over another.

📚 References

✔ Hasan, A., Hania, M., Mandall, N., & Fleming, P. S. (2023). The scientific evidence for pre-adjusted edgewise attachments and mechanics. British Dental Journal, 235, 175–181. https://doi.org/10.1038/s41415-023-6064-6
✔ Kattner, P. F., & Schneider, B. J. (1993). Comparison of Roth appliance and standard edgewise appliance treatment results. American Journal of Orthodontics and Dentofacial Orthopedics, 103(1), 24–32. https://doi.org/10.1016/0889-5406(93)70100-3
✔ Moesi, B., Dyer, F., & Benson, P. E. (2013). Roth versus MBT: Does bracket prescription have an effect on the subjective outcome of pre-adjusted edgewise treatment? European Journal of Orthodontics, 35(2), 236–243. https://doi.org/10.1093/ejo/cjr126
✔ Papageorgiou, S. N., Konstantinidis, I., Papadopoulou, K., Jäger, A., & Bourauel, C. (2014). Clinical effects of pre-adjusted edgewise orthodontic brackets: A systematic review and meta-analysis. European Journal of Orthodontics, 36(3), 350–363. https://doi.org/10.1093/ejo/cjt064
✔ Papageorgiou, S. N., Gkantidis, N., & Eliades, T. (2017). Treatment effects of various prescriptions and techniques for fixed orthodontic appliances: A systematic review. European Journal of Orthodontics, 39(1), 1–12.
✔ Roth, R. H. (1981). Functional occlusion for the orthodontist. Journal of Clinical Orthodontics, 15(1), 32–40, 44–51.
✔ Roth, R. H., & Rolfs, D. A. (1981). Functional occlusion for the orthodontist. Part II. Journal of Clinical Orthodontics, 15(2), 100–123.

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