Herpangina in Children: Causes, Symptoms, and Treatment of This Viral Infection

Herpangina is a common viral illness that primarily affects children under the age of five. It is marked by a sudden onset of fever, sore throat, and small ulcers or blisters in the back of the mouth—typically on the soft palate, uvula, and tonsils.

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Although self-limiting, early recognition is crucial to manage symptoms and prevent complications such as dehydration.

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✅ What Is Herpangina?
Herpangina is an acute viral infection characterized by painful mouth ulcers and systemic symptoms such as fever and malaise. It typically affects the posterior region of the oral cavity and is most prevalent during summer and early fall in the United States.

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✅ Causes (Etiology)
Herpangina is caused primarily by Coxsackievirus A, though other enteroviruses like Coxsackie B and echoviruses may also be responsible. These viruses spread easily through:

° Fecal-oral route
° Respiratory droplets
° Contaminated surfaces (e.g., toys, utensils)

Outbreaks are common in daycare centers and preschools (Khetsuriani et al., 2006).

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✅ Symptoms and Clinical Features
The condition develops rapidly and typically includes:

° High fever (101–104°F / 38.5–40°C)
° Sore throat and painful swallowing
° Loss of appetite
° Irritability
° Abdominal pain (occasionally)

➤ Oral findings appear within 24–48 hours:

° Small, fluid-filled blisters (1–2 mm) on the soft palate, uvula, and tonsils
° Blisters rupture into shallow ulcers with red halos
° Symptoms usually resolve in 5 to 7 days

Unlike hand, foot, and mouth disease, herpangina typically does not involve skin rashes or lesions on the hands and feet (Puenpa et al., 2019).

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✅ Differential Diagnosis
Conditions that may resemble herpangina include:

° Primary herpetic gingivostomatitis
° Strep throat (streptococcal pharyngitis)
° Infectious mononucleosis
° Hand-foot-and-mouth disease

Diagnosis is clinical and based on the child’s age, symptom pattern, and the appearance of the lesions. Lab tests are rarely needed.

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✅ Treatment
There is no specific antiviral medication for herpangina. Treatment focuses on supportive care:

➤ Hydration: Encourage frequent sips of water or electrolyte solutions
➤ Pain relief: Acetaminophen or ibuprofen for fever and sore throat
➤ Soft, cold foods: Popsicles, smoothies, and yogurt to reduce discomfort
➤ Avoid acidic or spicy foods: These may worsen oral pain

Antibiotics are not effective and should not be used unless there is a confirmed secondary bacterial infection.

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✅ Prevention
Key preventive strategies include:

° Frequent handwashing with soap and water
° Avoiding the sharing of utensils or cups
° Disinfecting surfaces and toys
° Keeping infected children home during the contagious period

There is no vaccine specifically for herpangina, though vaccine research targeting certain enteroviruses is ongoing in high-incidence regions.

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💬 Discussion
While herpangina is generally mild, it can be distressing for both children and parents due to painful symptoms and feeding difficulties. In some cases—especially in younger children—dehydration may require medical attention. Additionally, misdiagnosis can lead to inappropriate use of antibiotics, contributing to antibiotic resistance.
Public health education for caregivers and accurate clinical guidance are essential to avoid unnecessary treatments and improve patient outcomes. Healthcare providers should offer clear instructions for at-home care and signs that warrant medical evaluation.

💡 Conclusion
Herpangina is a self-limiting viral illness in children caused by enteroviruses such as Coxsackievirus A. It presents with fever, sore throat, and ulcers in the back of the mouth. Management is supportive, focusing on hydration and pain relief. Understanding its symptoms, transmission, and proper care helps reduce complications and prevents unnecessary medical interventions.

📚 References

✔ Khetsuriani, N., Lamonte-Fowlkes, A., Oberst, S., & Pallansch, M. A. (2006). Enterovirus surveillance—United States, 1970–2005. MMWR Surveillance Summaries, 55(8), 1–20. https://www.cdc.gov/mmwr/preview/mmwrhtml/ss5508a1.htm

✔ Puenpa, J., Vongpunsawad, S., & Poovorawan, Y. (2019). Enterovirus infections in children with herpangina and hand, foot, and mouth disease in Thailand, 2012–2018. Virology Journal, 16(1), 1–10. https://doi.org/10.1186/s12985-019-1202-0

✔ National Institutes of Health. (2022). Herpangina. MedlinePlus Medical Encyclopedia. https://medlineplus.gov/ency/article/001366.htm

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Why Does Diabetes Cause Dry Mouth? Understanding the Link Between Xerostomia and Blood Sugar Levels

Dry mouth, or xerostomia, is a common but often overlooked complication of diabetes mellitus. Affecting both type 1 and type 2 diabetes patients, this condition results from altered salivary gland function, often exacerbated by high blood glucose levels.

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Since saliva plays a critical role in maintaining oral and systemic health, understanding the mechanisms behind xerostomia in diabetes is essential for prevention and management of related complications.

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✅ The Role of Saliva in Oral Health
Saliva is crucial for maintaining oral homeostasis. It lubricates the oral tissues, aids in digestion, neutralizes acids, and has antimicrobial properties that protect against infections and tooth decay. A decrease in saliva flow or a change in its composition can disrupt this balance, leading to:

° Increased risk of dental caries
° Oral infections, such as candidiasis
° Burning mouth sensation
° Difficulty speaking, chewing, and swallowing

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✅ How Diabetes Leads to Dry Mouth

1. Hyperglycemia and Fluid Loss
Persistent high blood glucose levels lead to osmotic diuresis—a process in which glucose is excreted in the urine along with large amounts of water. This results in systemic dehydration, which also affects the salivary glands. Dehydration reduces salivary output and increases oral dryness (Lopez-Pintor et al., 2016).

2. Neuropathy Affecting Salivary Glands
Diabetic neuropathy, a common long-term complication of poorly controlled diabetes, can damage the autonomic nerves responsible for salivary gland stimulation. This autonomic dysfunction leads to reduced salivary secretion and altered gland response (Darwazeh & Al-Dwairi, 2019).

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3. Microvascular Damage
Diabetes can impair blood flow due to damage in the small blood vessels (microangiopathy), including those that supply the salivary glands. Reduced perfusion limits the glands' ability to function properly, contributing to xerostomia (Ghezzi & Ship, 2003).

4. Medication Side Effects
Many people with diabetes are on multiple medications, including antihypertensives, antidepressants, and diuretics. These drugs are known to cause dry mouth as a side effect, compounding the issue (Scully, 2003).

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💬 Discussion
Xerostomia in diabetes is multifactorial and often worsens with poor glycemic control. The consequences are not limited to discomfort; dry mouth can significantly impair a patient's quality of life and lead to further systemic complications. For instance, diabetic patients with xerostomia are more likely to develop periodontal disease, which in turn can increase systemic inflammation and complicate blood sugar management.
Healthcare professionals—including endocrinologists, primary care physicians, and dentists—must work collaboratively to identify and manage xerostomia early. Glycemic control should be the cornerstone of treatment, alongside patient education, salivary substitutes, sugar-free lozenges, and regular dental evaluations.

💡 Conclusion
Dry mouth is a common and clinically significant symptom in patients with diabetes. It results primarily from dehydration, neuropathy, vascular damage, and medication use. Effective management requires a comprehensive, multidisciplinary approach centered on optimal blood sugar control and targeted oral care strategies. Awareness of this connection can improve both oral and overall health outcomes in diabetic populations.

📚 References

✔ Darwazeh, A. M. G., & Al-Dwairi, Z. N. (2019). The relationship between xerostomia and glycemic control in patients with type 2 diabetes mellitus. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, 127(5), 409–416. https://doi.org/10.1016/j.oooo.2018.12.013

✔ Ghezzi, E. M., & Ship, J. A. (2003). Aging and secretory reserve capacity of major salivary glands. Journal of Dental Research, 82(10), 844–848. https://doi.org/10.1177/154405910308201106

✔ Lopez-Pintor, R. M., Casañas, E., González-Serrano, J., Serrano, J., & Hernández, G. (2016). Xerostomia, hyposalivation, and salivary flow in diabetes patients. Journal of Diabetes Research, 2016, 4372852. https://doi.org/10.1155/2016/4372852

✔ Scully, C. (2003). Drug effects on salivary glands: dry mouth. Oral Diseases, 9(4), 165–176. https://doi.org/10.1034/j.1601-0825.2003.03967.x

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Medications and Developing Teeth: Dental Risks, Mechanisms, and Prevention in Children

Tooth development is a complex process influenced by genetic and environmental factors, including exposure to certain medications. During critical stages—from pregnancy through early childhood—various drugs can interfere with odontogenesis, leading to permanent changes in tooth color, structure, and eruption patterns.

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Understanding how specific medications affect dental development is crucial for pediatricians, dentists, and caregivers to make informed decisions and prevent long-term oral health issues.

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✅ Dental Development and Critical Windows
Odontogenesis begins around the 6th to 8th week of gestation and continues into adolescence. The most vulnerable phases include:

➤ Amelogenesis: enamel formation.
➤ Dentinogenesis: dentin formation.
➤ Calcification and eruption: mineralization and emergence of the tooth into the oral cavity.

Cells like ameloblasts and odontoblasts are especially sensitive to systemic disturbances during these stages.

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✅ Medications Commonly Linked to Dental Effects

1. Tetracyclines
Tetracyclines (e.g., doxycycline, tetracycline) bind to calcium ions and become incorporated into developing dentin and enamel, causing yellow to brown tooth discoloration and enamel hypoplasia. These antibiotics are contraindicated in children under age 8 and during pregnancy (Chopra & Roberts, 2020).

2. Excessive Fluoride
Prolonged intake of fluoride above recommended levels—whether from supplements, toothpaste, or water—can lead to dental fluorosis. This enamel defect ranges from mild white streaks to severe brown staining and surface irregularities (Wong et al., 2011).

3. Sugary Syrups, Antihistamines, and Asthma Medications
Pediatric medications often come in syrup forms with high sugar content. Chronic use increases the risk of early childhood caries. Additionally, some antihistamines and bronchodilators reduce salivary flow, contributing to enamel demineralization and increased caries risk (Daly et al., 2021).

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4. Chemotherapy and Radiation Therapy in Pediatric Patients
Cancer treatments during childhood can disrupt tooth development, leading to enamel hypoplasia, microdontia, delayed eruption, or root malformations. The younger the child at the time of therapy, the greater the impact (Pérez et al., 2019).

5. Teratogenic Drugs: Thalidomide and Anticonvulsants
Drugs like thalidomide, known for causing congenital abnormalities, may result in craniofacial defects and missing teeth. Phenytoin, an anticonvulsant, is associated with gingival overgrowth and abnormal tooth eruption patterns (Naziri et al., 2022).

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💬 Discussion
Tooth development is highly sensitive to pharmacological interference. The consequences of early exposure to certain drugs are not only cosmetic but also functional—affecting chewing, speech, and a child’s self-esteem. Preventive efforts must prioritize careful medication prescribing during pregnancy and childhood, use of sugar-free formulations, and regular dental monitoring.
Healthcare providers should work collaboratively across disciplines—medical, dental, and pharmaceutical—to reduce the risks. Early oral health education for caregivers is equally important to ensure safe medication practices and early detection of developmental dental problems.

💡 Conclusion
Several medications can cause permanent changes in tooth development when administered during critical periods. Avoiding high-risk drugs in pregnancy and early childhood, choosing sugar-free options, and ensuring regular dental follow-up are key strategies for prevention. Coordinated care and caregiver awareness play essential roles in protecting pediatric oral health.

📚 References

✔ Chopra, I., & Roberts, M. (2020). Tetracycline antibiotics: mode of action, applications, molecular biology, and epidemiology of bacterial resistance. Microbiology and Molecular Biology Reviews, 65(2), 232–260. https://doi.org/10.1128/MMBR.65.2.232-260.2001

✔ Daly, B., Thompsell, A., Rooney, Y. M., & White, D. A. (2021). Oral health and drug therapy in children: a review. British Dental Journal, 231(4), 225–230. https://doi.org/10.1038/s41415-021-2913-7

✔ Naziri, E., Karami, E., & Torabzadeh, H. (2022). The effect of antiepileptic drugs on oral health in pediatric patients. Journal of Pediatric Dentistry, 10(1), 45–50. https://doi.org/10.1055/s-0042-1742451

✔ Pérez, J. R., Luján, A., & Moraes, A. (2019). Dental abnormalities after pediatric cancer therapy: clinical considerations. Pediatric Dentistry Journal, 44(2), 89–96. https://doi.org/10.1016/j.pdj.2018.09.003

✔ Wong, M. C. M., Glenny, A. M., Tsang, B. W. Y., Lo, E. C. M., Worthington, H. V., & Marinho, V. C. C. (2011). Topical fluoride for caries prevention in children and adolescents. Cochrane Database of Systematic Reviews, (1). https://doi.org/10.1002/14651858.CD007693.pub2

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How to Correct Harmful Oral Habits in Children That Affect Facial and Dental Development

Early childhood is a critical period for craniofacial and dental development. Certain harmful oral habits, such as thumb sucking, mouth breathing, or nail biting, can interfere with proper facial growth and tooth alignment.

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If not addressed early, these habits may lead to malocclusion, facial asymmetry, and the need for complex orthodontic treatment later in life. This article outlines the most common harmful oral habits in children, their effects on dental and facial development, and effective evidence-based treatment strategies.

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✅ Common Harmful Oral Habits in Children: Definitions and Treatments

1. Thumb Sucking
➤ Definition:
° A repetitive behavior in which the child inserts one or more fingers into the mouth, usually for comfort or stress relief.
➤ Potential Effects:
° Anterior open bite
° Protrusion of upper front teeth
° Underdeveloped lower jaw
° Improper lip seal
➤ Treatment Options:
° Positive reinforcement techniques (e.g., reward charts)
° Behavior tracking with family support
° Intraoral appliances (e.g., palatal crib or tongue rake) in persistent cases
° Psychological support for anxiety-linked cases (Barbería et al., 2021)

2. Prolonged Pacifier or Bottle Use
➤ Definition:
° Using a pacifier or bottle beyond age 2–3, leading to non-nutritive sucking behavior.
➤ Potential Effects:
° Anterior open bite
° Posterior crossbite
° High, narrow palate
➤ Treatment Options:
° Parent education on weaning by age 2
° Gradual transition to cups and comfort objects
° Orthodontic intervention if malocclusion persists
° Oral muscle training to improve lip seal and tongue posture

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3. Tongue Thrust (Atypical Swallowing)
➤ Definition:
° Pushing the tongue against or between the teeth when swallowing or speaking, instead of placing it against the palate.
➤ Potential Effects:
° Anterior open bite
° Gaps between front teeth
° Weak orofacial muscles
➤ Treatment Options:
° Orofacial myofunctional therapy (OMT)
° Palatal cribs or tongue spurs if habit continues past age 6
° Collaboration with a speech-language pathologist
° Long-term monitoring by pediatric dentist or orthodontist

4. Mouth Breathing
➤ Definition:
° Breathing through the mouth instead of the nose, often due to nasal obstruction or habit.
➤ Potential Effects:
° Long face syndrome
° Incompetent lips (open mouth posture)
° Narrow upper jaw and posterior crossbite
° High-arched palate
➤ Treatment Options:
° ENT evaluation for nasal or adenoid obstruction
° Palatal expansion in cases of narrow maxilla
° Orofacial therapy to re-establish nasal breathing
° Nasal hygiene and breathing retraining exercises

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5. Nail Biting (Onychophagia)
➤ Definition:
° A compulsive habit of biting or chewing nails, often triggered by stress or anxiety.
➤ Potential Effects:
° Tooth wear or misalignment
° Microfractures in front teeth
° Risk of infections around the mouth
° Jaw tension or muscle strain
➤ Treatment Options:
° Behavioral strategies (e.g., bitter nail polish, habit reversal training)
° Psychological support if anxiety-related
° Orofacial therapy to manage perioral muscle tension
° Parental coaching and support at home and school (Maia et al., 2019)

✅ Diagnosis
A thorough diagnosis involves both physical and behavioral evaluation:
Comprehensive dental and facial exam
History of the habit (age of onset, frequency, triggers)
Functional assessment of breathing, swallowing, and oral posture
Referral to ENT, speech therapist, or child psychologist if needed

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💬 Discussion
Persistent oral habits beyond ages 3–4 can significantly impact a child's bite, facial symmetry, and speech development. Studies have shown that early intervention is key, ideally before age 6, when craniofacial structures are still adaptable (Grippaudo et al., 2020; Souki et al., 2019).
Most habits can be addressed successfully through behavioral therapy and parent involvement. In more severe cases, interceptive orthodontics or interdisciplinary care may be required. Educating caregivers is essential for consistent support at home.

💡 Conclusion
Harmful oral habits can disrupt normal facial and dental development if not treated in time. Each habit presents specific risks and requires a tailored treatment approach. Early identification, behavioral guidance, and, when necessary, interdisciplinary therapy, offer the best outcomes. Prevention and early parental education remain the most effective tools in managing these behaviors.

📚 References

✔ Barbería, E., Lucavechi, T., & Suárez-Clúa, M. C. (2021). Clinical Pediatric Dentistry. Elsevier España.

✔ Grippaudo, C., Paolantonio, E. G., Antonini, G., Saulle, R., La Torre, G., & Deli, R. (2020). Association between oral habits, mouth breathing and malocclusion. Acta Otorhinolaryngologica Italica, 40(5), 282–289. https://doi.org/10.14639/0392-100X-N0616

✔ Souki, B. Q., Pimenta, G. B., Souki, M. Q., Franco, L. P., Becker, H. M. G., & Pinto, J. A. (2019). Prevalence of malocclusion among mouth breathing children: do expectations meet reality? International Journal of Pediatric Otorhinolaryngology, 119, 146–150. https://doi.org/10.1016/j.ijporl.2019.01.032

✔ Viggiano, D., Fasano, D., Monaco, G., & Strohmenger, L. (2020). Oral habits and orthodontic anomalies in preschool children. International Journal of Paediatric Dentistry, 30(3), 326–333. https://doi.org/10.1111/ipd.12594

✔ Maia, B. R., Marques, D. R., & Barbosa, F. (2019). Nail biting in children: an integrative review. Psicologia: Reflexão e Crítica, 32(1), 1–9. https://doi.org/10.1186/s41155-019-0116-1

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Myofascial Pain Syndrome in Dentistry: Clinical Impact and Modern Management

Orofacial pain is a common concern in dental practice and may stem from various sources including dental, joint, neuropathic, or muscular origins.

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Myofascial pain syndrome (MPS) is one of the most prevalent muscular causes of orofacial and jaw pain, often presenting with facial trigger points and mimicking temporomandibular joint dysfunction (TMJ disorder) or tooth pain. Early recognition is key to avoiding misdiagnosis and unnecessary dental procedures.

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✅ Definition of Myofascial Pain Syndrome
MPS is defined as a chronic pain condition involving myofascial trigger points—hyperirritable spots located within taut bands of skeletal muscle or fascia. When palpated, these points produce localized or referred pain and muscular stiffness (Simons et al., 1999; Gerwin, 2020).

✅ Causes and Risk Factors
Several factors contribute to the onset of MPS, including:

° Bruxism and chronic jaw clenching
° Muscle overuse (e.g., prolonged chewing, poor posture)
° Emotional stress, leading to muscle tension
° Poor occlusion or dental misalignment
° Tooth loss or unbalanced prosthetics
° Jaw trauma or repetitive microtrauma

These triggers can result in dysfunctional muscle contraction and sustained activation of trigger points.

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✅ Symptoms and Clinical Presentation
Typical symptoms of MPS in dental patients include:

° Persistent jaw pain or soreness
° Facial muscle stiffness and tenderness
° Referred pain to the teeth, temples, ears, or neck
° Jaw fatigue or tightness during talking or chewing
° Clicking or limited range of motion in the TMJ
° Sensation of malocclusion without clinical evidence

These symptoms often resemble TMD or neuropathic conditions, making clinical evaluation essential.

✅ Clinical Relevance in Dentistry
Myofascial pain can complicate dental diagnosis and management due to symptom overlap with:

° Atypical toothache (non-odontogenic pain)
° Temporomandibular joint disorders (TMJ/TMD)
° Facial nerve pain or neuralgia
° Persistent post-treatment dental pain

According to the American Academy of Orofacial Pain (2022), up to 85% of patients with chronic orofacial pain have a muscular component. Failing to recognize MPS can result in unnecessary root canals, extractions, or surgical interventions.

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✅ Diagnosis
MPS diagnosis is clinical and based on:

° Palpation of active trigger points
° Reproduction of referred pain
° Muscle tightness and tenderness
° Exclusion of dental or joint pathologies

Imaging is not typically required but may be helpful in ruling out other causes. Ultrasound and electromyography are sometimes used for muscle assessment.

✅ Treatment Options
Effective management of MPS is multidisciplinary and includes:

➤ Physical Therapy and Manual Techniques
°Myofascial release and massage therapy
° Dry needling
° Jaw stretching and strengthening exercises
° Ultrasound therapy or heat application

➤ Medications
° Nonsteroidal anti-inflammatory drugs (NSAIDs)
° Muscle relaxants (e.g., cyclobenzaprine)
° Local anesthetic injections for trigger point relief

➤ Dental Management
° Occlusal adjustments and bite correction
° Night guards or splint therapy for bruxism
° Replacement of missing teeth to restore occlusal balance

➤ Psychological and Behavioral Support
° Cognitive-behavioral therapy for stress and anxiety
° Biofeedback or relaxation techniques

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💡 Conclusion
Myofascial pain syndrome is a common yet frequently overlooked source of jaw muscle pain and orofacial dysfunction in dental patients. Its overlapping symptoms with other dental and TMJ conditions can lead to misdiagnosis and overtreatment. Dentists must be equipped to recognize the signs of MPS and apply integrated approaches involving manual therapy, medication, and occlusal management. Early diagnosis is critical for successful pain relief and restoration of normal function.

📚 References

✔ American Academy of Orofacial Pain. (2022). Orofacial Pain: Guidelines for Assessment, Diagnosis, and Management (6th ed.). Quintessence Publishing.

✔ Gerwin, R. D. (2020). Classification, epidemiology, and etiology of myofascial pain syndrome. Current Pain and Headache Reports, 24(5), 1–6. https://doi.org/10.1007/s11916-020-00832-5

✔ Simons, D. G., Travell, J. G., & Simons, L. S. (1999). Myofascial Pain and Dysfunction: The Trigger Point Manual (2nd ed.). Williams & Wilkins.

✔ Fernández-de-Las-Peñas, C., & Dommerholt, J. (2018). Myofascial Trigger Points: Pathophysiology and Evidence-Informed Diagnosis and Management. Jones & Bartlett Learning.

✔ Manfredini, D., & Guarda-Nardini, L. (2020). Myofascial pain in temporomandibular disorders: An updated review on diagnosis and management. Journal of Oral Rehabilitation, 47(5), 670–682. https://doi.org/10.1111/joor.12930

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Oral Dermatitis Treatment: A Complete Guide for Dentists in 2025

Oral dermatitis, though relatively uncommon, presents significant diagnostic and therapeutic challenges for dental professionals.

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With its multifactorial etiology—ranging from contact allergies to systemic diseases—accurate recognition and appropriate intervention are critical. This article explores the current understanding, dental management, and evidence-based treatments for oral dermatitis in 2025.

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✅ Introduction
Oral dermatitis, often referred to as perioral dermatitis when it involves the skin around the mouth, can extend to intraoral tissues and is sometimes misdiagnosed as other mucosal conditions. Dentists play a vital role in recognizing these presentations, especially as many triggers involve materials used in dental practice, such as resins, toothpaste ingredients, or latex.

✅ Etiology and Pathophysiology
Oral dermatitis may stem from various sources:

° Contact allergens: toothpaste (especially those with sodium lauryl sulfate), cinnamon flavorings, dental materials (e.g., methacrylates), or latex gloves.
° Topical corticosteroids: Ironically, overuse of facial topical steroids can trigger perioral dermatitis, including intraoral symptoms.
° Microbial factors: Overgrowth of Candida albicans or bacterial dysbiosis.
° Systemic diseases: Lupus erythematosus, Crohn's disease, or atopic dermatitis.

Histologically, oral dermatitis involves spongiotic mucositis with lymphocytic infiltration and variable epithelial degeneration.

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✅ Clinical Presentation
Symptoms commonly include:

° Erythema and desquamation around lips and perioral skin
° Burning sensation, especially after eating spicy or acidic foods
° Intraoral white patches or erythematous areas, sometimes mistaken for candidiasis
° Dryness, cracking, or angular cheilitis

These signs may fluctuate and overlap with other conditions such as oral lichen planus, allergic stomatitis, or cheilitis granulomatosa.

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✅ Diagnosis

➤ Step-by-step evaluation by the dentist:
1. Detailed history: Include dietary habits, oral hygiene products, recent dental restorations, and use of topical medications.
2. Clinical examination: Note distribution of lesions, mucosal texture, and presence of vesicles or ulcers.
3. Allergy testing: Patch tests or ELISA to identify specific allergens.
4. Biopsy: In persistent or unclear cases, to exclude autoimmune or neoplastic conditions.
5. Microbial culture: Especially to rule out superinfection by Candida or bacteria.

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✅ Dental Management Strategies

➤ Elimination of Irritants
° Advise discontinuation of products with sodium lauryl sulfate, triclosan, or strong flavorings.
° Switch to hypoallergenic or fluoride-free toothpaste if needed.

➤ Topical Therapy
° Non-fluorinated topical calcineurin inhibitors (e.g., tacrolimus 0.03%) have shown efficacy (Wollenberg et al., 2023).
° Antifungals if Candida is detected (e.g., miconazole gel).

➤ Oral Rinse Regimen
° Alcohol-free chlorhexidine or baking soda rinses to reduce microbial load and irritation.

➤ Nutritional Support
° Vitamin B complex and iron supplements if deficiencies are noted.

➤ Restorative Material Evaluation
° Replace suspected allergenic materials with biocompatible alternatives (e.g., zirconia, BPA-free composites).

➤ Patient Education
° Emphasize gentle oral hygiene, avoiding abrasive brushing or mouthwashes.
° Warn against unsupervised use of topical corticosteroids.

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💬 Discussion

The role of dentists in recognizing and managing oral dermatitis is growing, especially as cosmetic and restorative dental procedures increase in the U.S. The overlap with dermatologic and autoimmune conditions necessitates interdisciplinary collaboration. Early intervention not only prevents chronic mucosal damage but also improves patient quality of life. Furthermore, dentists should remain aware of trends in consumer products and materials that may trigger these reactions.

💡 Conclusion

Oral dermatitis remains a complex, underdiagnosed condition requiring careful assessment by dental professionals. By integrating updated diagnostic protocols, biocompatible materials, and patient-specific management strategies, dentists can effectively treat this condition while minimizing recurrence.

📚 References

✔ Wollenberg, A., Barbarot, S., Bieber, T., Christen-Zaech, S., Deleuran, M., Fölster-Holst, R., ... & Cork, M. (2023). European Dermatology Forum guideline on topical calcineurin inhibitors in dermatology, 2023 update. Journal of the European Academy of Dermatology and Venereology, 37(4), 675–686. https://doi.org/10.1111/jdv.19034

✔ Rantanen, T., & Jolanki, R. (2021). Allergic reactions to dental materials. Contact Dermatitis, 84(3), 145–152. https://doi.org/10.1111/cod.13839

✔ Van Hoogstraten, I. M., et al. (2020). Oral mucosal contact allergies: Recognition, diagnosis, and management. Clinical Oral Investigations, 24(8), 2641–2650. https://doi.org/10.1007/s00784-020-03288-3

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Risk Factors, Signs & Modern Management of Tongue Cancer: 2025 Review

Tongue cancer—primarily squamous cell carcinoma (SCC)—represents a significant portion of head and neck malignancies. Although relatively rare (less than 1 % of new cancer cases), its incidence is rising among younger, non‑smoking populations .

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This review synthesizes risk factors, clinical presentation, diagnostic pathways, treatment modalities, and preventive strategies over the past six years.

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✅ Risk Factors

° Tobacco smoking and smokeless tobacco: Primary risk factor; smoking increases risk 3–6× and smokeless forms similarly elevate risk.
° Alcohol consumption: Heavy alcohol use independently increases risk (~20 % of oral cancers), synergizes with tobacco to cause ~72 % of head and neck cancers.
° HPV infection: High-risk strains (HPV‑16/18) are strongly linked to base‑of‑tongue SCC; rising HPV-driven oropharyngeal cancers.
° Betel nut and paan chewing: Doubles oral cancer risk even without tobacco, contributing up to 40 % of cases in endemic regions.
° Chronic inflammation and oral dysbiosis: Emerging research links chronic oral irritation/infections with oxidative stress and carcinogenesis.
° Non-modifiable factors: Includes age (≥ 40), male sex (2× risk), race (higher in African‑American men), family or personal cancer history.

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✅ Signs & Symptoms

° Non-healing ulcers or sores, red/white patches (erythroplakia, leukoplakia).
° Lumps or thickening on tongue; easy bleeding.
° Pain, burning, numbness, or discomfort in mouth/jaw .
° Functional issues: difficulty chewing, swallowing, speaking; ear, throat, or jaw pain; loose teeth .
° Late-stage signs: neck lymphadenopathy, weight loss, bleeding, voice changes.

✅ Diagnosis

° Clinical exam: Visual and manual inspection, mirror or endoscopic (nasendoscopy) evaluation.
° Biopsy & histopathology: Essential for definitive diagnosis. HPV testing often included.
° Imaging: CT, MRI, ultrasound, PET‑CT to assess tumor extent and lymph node involvement.

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✅ Treatment

° Surgery: Primary for early-stage disease; transoral resections for oral tongue; neck dissection as needed.
° Radiation therapy: Often adjuvant post-surgery or primary modality for advanced/inoperable cases.
° Chemotherapy & chemoradiation: Platinum-based regimens, especially for HPV-negative or bulky disease .
° Targeted therapies & immunotherapy: Emerging modalities; microenvironment-targeted immunotherapy under study.
° Supportive care: Speech/swallow rehabilitation, nutritional support to manage side effects.

✅ Prevention

° Smoking cessation and reduced alcohol intake are the most effective strategies.
° HPV vaccination (recommended through age 45) may reduce HPV-driven tongue cancer.
° Avoid betel nut/paan; improve oral hygiene and diet (fruit/vegetable intake).
° Regular dental/oral screenings with prompt evaluation of lesions persisting >2–3 weeks .

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💬 Discussion

Recent epidemiological trends indicate an increase in tongue cancer among young, non-smoking individuals, highlighting the growing importance of HPV and alternative risk factors. Concurrently, advancements in diagnostic imaging, minimally invasive surgical techniques, and immunotherapies offer improved outcomes and functional preservation. However, challenges persist: late-stage presentation, therapy-related toxicity, and disparities in access to HPV vaccination and specialist care.

💡 Conclusion

Tongue cancer is a multifactorial disease shaped by lifestyle, viral, environmental, and biological determinants. Contemporary understanding underscores the interplay of traditional risks—tobacco and alcohol—with newer drivers like HPV, chronic inflammation, and betel nut use. Early detection and a multidisciplinary treatment approach remain paramount. Preventive measures, especially smoking cessation, alcohol reduction, and HPV vaccination, are critical for reducing incidence.

📚 References

✔ American Cancer Society. (2021). Risk Factors for Oral Cavity and Oropharyngeal Cancers. Retrieved from American Cancer Society website.

✔ Cleveland Clinic. (2023). Tongue cancer: symptoms, causes & treatment. Cleveland Clinic.

✔ Mayo Clinic Staff. (2025, November). Tongue cancer: symptoms and causes. Mayo Clinic.

✔ Current trends on prevalence, risk factors and prevention of oral... Frontiers in Oral Health, 2024.

✔ Exploration Pub. (2024). Risk factors, prevention, diagnosis, and immunotherapy’s role in oral cancer.

✔ Verywell Health. (2024, April 22). Tongue Cancer: Everything You Need to Know.

✔ New York Post. (2025, May). Top Chef alum Shirley Chung cancer update.

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Common Complications of Dental Implants: Diagnosis and Treatment Guide

Dental implants have become the gold standard for replacing missing teeth due to their high success rate and ability to restore function and aesthetics. However, like any surgical procedure, implant placement is not free of complications.

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Understanding the nature of these complications—ranging from peri-implant diseases to mechanical failures—is crucial for timely diagnosis and appropriate management. This article reviews the most common problems associated with dental implants, including their definitions, clinical characteristics, diagnostic strategies, and current treatment options.

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✅ 1. Peri-Implant Mucositis

➤ Definition:
° Peri-implant mucositis is a reversible inflammatory reaction of the soft tissues surrounding a dental implant without accompanying bone loss.
➤ Clinical Features:
° Redness and swelling of peri-implant mucosa
° Bleeding on probing (BOP)
° No radiographic bone loss
° Patient may report mild discomfort or sensitivity
➤ Diagnosis:
° Probing depth measurement
° Presence of BOP
° Radiographs confirm absence of bone loss
° Exclusion of other causes such as food impaction or residual cement
➤ Treatment:
° Mechanical debridement with plastic or titanium curettes
° Antimicrobial mouth rinses (e.g., chlorhexidine)
° Improved patient oral hygiene
° Re-evaluation after 2–4 weeks

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✅ 2. Peri-Implantitis

➤ Definition:
° Peri-implantitis is a progressive inflammatory disease affecting both the soft and hard tissues around an osseointegrated implant, leading to bone loss.
➤ Clinical Features:
° BOP and/or suppuration
° Increased probing depth (>5 mm)
° Progressive radiographic bone loss
° Possible implant mobility in advanced cases
➤ Diagnosis:
° Periodontal charting (baseline comparison)
° Radiographic bone level analysis
° Microbial analysis in severe or refractory cases
➤ Treatment:
° Mechanical debridement and antiseptic therapy
° Local or systemic antibiotics (e.g., amoxicillin + metronidazole)
° Surgical intervention (e.g., resective or regenerative surgery)
° Implant surface decontamination with lasers or air abrasives

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✅ 3. Implant Failure (Early and Late)

➤ Definition:
° Implant failure is classified as early (before osseointegration) or late (after functional loading), resulting in implant mobility or loss.
➤ Clinical Features:
° Pain or discomfort on function
° Mobility of implant
° Radiographic evidence of peri-implant radiolucency
° Soft tissue inflammation
➤ Diagnosis:
° Clinical mobility testing
° Percussion and tactile evaluation
° Radiographs to assess integration and bone levels
➤ Treatment:
° Removal of failed implant
° Management of infection or bone defects
° Possible delayed or immediate re-implantation depending on case

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✅ 4. Mechanical Complications

a. Screw Loosening or Fracture
➤ Definition:
° Mechanical dislodgment or breakage of abutment or prosthetic screws.
➤ Clinical Features:
° Mobility of crown or prosthesis
° Clicking or instability during function
° Possible pain or soft tissue trauma
➤ Diagnosis:
° Clinical inspection
° Radiographs to detect screw fracture or misfit
➤ Treatment:
° Retightening or replacement of screws
° Use of torque-controlled drivers
° Avoidance of occlusal overload

b. Prosthetic Fracture (e.g., Crown or Bridge)
➤ Definition:
° Fracture of the prosthetic components due to stress, fatigue, or poor design.
➤ Clinical Features:
° Fractured ceramic or acrylic visible
° Aesthetic compromise
° Patient may report altered bite or discomfort
➤ Diagnosis:
° Clinical examination
° Assessment of occlusal forces and design flaws
➤ Treatment:
° Repair or replacement of prosthesis
° Occlusal adjustment
° Use of more durable materials (e.g., zirconia)

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✅ 5. Neurological Complications

➤ Definition:
° Nerve injury typically occurs during implant placement in the mandibular region, resulting in temporary or permanent paresthesia.
➤ Clinical Features:
° Numbness or tingling of lower lip, chin, or tongue
° Burning sensation
° Pain or discomfort during healing
➤ Diagnosis:
° Clinical sensory testing (light touch, pinprick)
° Radiographic assessment of implant proximity to nerve canal
° Cone beam computed tomography (CBCT) if needed
➤ Treatment:
° Immediate implant removal if impingement is suspected
° Corticosteroids to reduce inflammation
° Referral to a neurologist for persistent symptoms

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💬 Discussion
Dental implant complications can significantly impact treatment outcomes and patient satisfaction. Peri-implant diseases, including mucositis and peri-implantitis, are among the most common biological complications and share many features with periodontal diseases. Mechanical and neurological complications, although less frequent, require early recognition and targeted management. Preventive strategies such as accurate surgical planning, patient education, and regular maintenance therapy play a vital role in minimizing the occurrence of complications.

💡 Conclusion
While dental implants are highly predictable, complications—both biological and mechanical—can arise. Timely identification and management based on clinical and radiographic findings are essential for preserving implant function and health. Clinicians must stay updated on the latest diagnostic protocols and treatment strategies to ensure long-term success and patient safety.

📚 References

✔ Lang, N. P., Berglundh, T., & Working Group 4 of the Seventh European Workshop on Periodontology. (2011). Periimplant diseases: Where are we now? – Consensus of the Seventh European Workshop on Periodontology. Journal of Clinical Periodontology, 38(s11), 178–181. https://doi.org/10.1111/j.1600-051X.2010.01674.x

✔ Heitz-Mayfield, L. J., & Mombelli, A. (2014). The therapy of peri-implantitis: A systematic review. The International Journal of Oral & Maxillofacial Implants, 29(Suppl), 325–345. https://doi.org/10.11607/jomi.2014suppl.g5.3

✔ Esposito, M., Hirsch, J. M., Lekholm, U., & Thomsen, P. (1998). Biological factors contributing to failures of osseointegrated oral implants. (I). Success criteria and epidemiology. European Journal of Oral Sciences, 106(1), 527–551. https://doi.org/10.1046/j.0909-8836.1998.eos106111.x

✔ Misch, C. E. (2020). Dental Implant Prosthetics (3rd ed.). Mosby.

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Oral Manifestations of STDs: Diagnosis, Signs, and Dental Management

Sexually transmitted diseases (STDs) remain a global health concern. Several infections, including syphilis, HIV, herpes simplex virus (HSV), gonorrhea, and human papillomavirus (HPV), present oral signs that may be the first indication of systemic illness.

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Dentists are in a unique position to detect and refer these cases early.

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✅ Diagnosis in dental practice
A thorough medical and sexual history should be taken. Diagnostic tools include:

° Biopsy and exfoliative cytology
° PCR testing for viral identification (HPV, HSV)
° Serologic testing (VDRL, ELISA, Western Blot)
° Referral to specialists for confirmatory diagnosis

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✅ Common oral manifestations of STDs

✅ Dental management
A thoughtful and professional approach is essential when treating patients with oral manifestations of STDs. Management includes:

1. Thorough clinical examination:
° Detailed intraoral inspection to identify ulcers, warts, vesicles, leukoplakia, or other lesions.
° Clinical photography (with consent) for documentation and follow-up.
2. Detailed medical history:
° Focus on systemic conditions, sexual history, and risk behaviors (e.g., smoking, drug use, multiple partners).
3. Symptomatic management:
° Topical anesthetics or systemic analgesics for pain.
° Antiviral therapy (e.g., Acyclovir, Valacyclovir) for herpes infections.
° Systemic antibiotics for bacterial STDs (e.g., syphilis or gonorrhea)—in coordination with medical treatment.
° Antifungal therapy for HIV-related oral candidiasis (e.g., Nystatin, Fluconazole).
4. Patient education and counseling:
° Clarify the link between STDs and oral health.
° Promote safer sex practices and regular testing.
° Encourage communication with physicians and follow-up care.
5. Referral and interdisciplinary care:
° Timely referral to infectious disease specialists or physicians.
° Collaboration with medical teams for systemic management.

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✅ Basic biosafety protocols in dental practice
To ensure safety for both dental personnel and patients, the following standard precautions must be implemented:

➤ Personal Protective Equipment (PPE):
° Gloves, surgical mask or N95 respirator (if aerosol is generated), protective eyewear, and disposable gowns.
➤ Strict hand hygiene:
° Follow the WHO's five moments of hand hygiene protocol before and after patient contact.
➤ Surface and instrument sterilization:
° Proper cleaning and disinfection of all equipment and environmental surfaces between patients.
➤ Aerosol minimization:
° Use low-speed instruments or hand techniques when possible, especially with active lesions or immunocompromised patients.
➤ Biological waste management:
° Proper segregation and disposal of contaminated materials per regulations.
➤ Informed consent:
° Secure written consent before biopsy, photo documentation, or referral procedures.

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💡 Conclusion
Oral signs may be the first or only indication of STDs. Dentists must stay updated on their recognition and management to improve public health outcomes through early diagnosis and referral.

📚 References

✔ Fatahzadeh, M., & Schwartz, R. A. (2007). Human herpes simplex virus infections: epidemiology, pathogenesis, symptomatology, diagnosis, and management. Journal of the American Academy of Dermatology, 57(5), 737–763. https://doi.org/10.1016/j.jaad.2007.06.020

✔ Neville, B. W., Damm, D. D., Allen, C. M., & Chi, A. C. (2015). Oral and Maxillofacial Pathology (4th ed.). Elsevier Health Sciences.

✔ Centers for Disease Control and Prevention (CDC). (2023). Sexually Transmitted Infections Treatment Guidelines. https://www.cdc.gov/std/treatment-guidelines/default.htm

✔ Reznik, D. A. (2006). Oral manifestations of HIV disease. Topics in HIV Medicine, 14(5), 143–148. https://pubmed.ncbi.nlm.nih.gov/17133174/

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Oral Manifestations of Systemic Diseases: Updated Clinical Review

Systemic diseases affect more than internal organs—they often present oral signs that can be essential for early diagnosis.

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Due to its high vascularity and immune role, the oral cavity frequently reflects systemic conditions. In modern dentistry, recognizing these signs is key to preventive care and interdisciplinary treatment.

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✅ Systemic Diseases and Their Oral Manifestations

1. Diabetes Mellitus
➤ Definition: A chronic metabolic disorder characterized by persistent hyperglycemia.
➤ Oral manifestations:
° Xerostomia (dry mouth)
° Advanced periodontal disease
° Delayed wound healing
° Oral candidiasis
➤ Common signs and symptoms: Gingival bleeding, tooth mobility, halitosis, recurrent oral infections.

2. Iron Deficiency Anemia
➤ Definition: A lack of iron leading to decreased oxygen-carrying capacity of the blood.
➤ Oral manifestations:
° Pale oral mucosa
° Atrophic glossitis (smooth, sore tongue)
° Angular cheilitis
➤ Common signs and symptoms: Burning sensation in the mouth, altered taste, oral ulcers.

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3. HIV/AIDS
➤ Definition: Infection caused by the human immunodeficiency virus, impairing the immune system.
➤ Oral manifestations:
° Pseudomembranous candidiasis
° Kaposi's sarcoma
° Oral hairy leukoplakia
° Necrotizing gingivitis/periodontitis
➤ Common signs and symptoms: White lesions, persistent ulcers, oral pain, gingival bleeding.

4. Systemic Lupus Erythematosus (SLE)
➤ Definition: A multisystem autoimmune disease affecting connective tissues.
➤ Oral manifestations:
° Painless oral ulcers
° Erythematous or purpuric lesions
° Xerostomia (often secondary to Sjögren’s syndrome)
➤ Common signs and symptoms: Mild pain, difficulty chewing, mucosal and lingual changes.

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5. Chronic Kidney Disease (CKD)
➤ Definition: Progressive, irreversible loss of kidney function.
➤ Oral manifestations:
° Uremic breath odor
° Oral ulcerations
° Gingival bleeding
Enamel hypoplasia (especially in children)
➤ Common signs and symptoms: Metallic taste, oral pain, delayed tooth eruption in children.

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✅ Recommendations for Dental Practice

° Conduct thorough medical histories that screen for systemic illnesses.
° Treat oral signs as potential indicators of undiagnosed systemic disease.
° Work collaboratively with physicians for interdisciplinary care.
° Educate patients on the oral-systemic health connection.
° Schedule frequent cleanings and periodontal evaluations for at-risk patients.

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💡Conclusions
Oral signs of systemic diseases serve as critical indicators for early diagnosis and comprehensive treatment. Timely recognition of these signs can improve patient outcomes, reduce complications, and enhance quality of life. Dentists play a key role in identifying these manifestations and guiding patients to appropriate medical care.

📚 References

✔ Chávez, E. M., et al. (2022). Oral health considerations in patients with chronic kidney disease. Journal of Clinical Nephrology and Renal Care, 8(1), 110. https://doi.org/10.23937/2572-3286.1510110

✔ Kottoor, R., et al. (2020). Oral manifestations in patients with systemic lupus erythematosus. Lupus, 29(3), 274–281. https://doi.org/10.1177/0961203320903073

✔ López-López, J., et al. (2021). Oral manifestations of iron deficiency anemia: A clinical approach. Clinical Oral Investigations, 25(1), 123–130. https://doi.org/10.1007/s00784-020-03418-z

✔ López-Pintor, R. M., et al. (2020). Diabetes mellitus and oral health: A bidirectional relationship. Medicina Oral, Patología Oral y Cirugía Bucal, 25(4), e559–e566. https://doi.org/10.4317/medoral.24012

✔ Patton, L. L., et al. (2021). Oral manifestations of HIV infection and treatment: A clinical guide. Oral Diseases, 27(S1), 59–68. https://doi.org/10.1111/odi.13742

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Lingual Coating: Causes, Characteristics, Bacterial Profile, Consequences, and Treatment

Lingual coating, also known as tongue biofilm or tongue plaque, is a common condition in the oral cavity characterized by a whitish, yellowish, or brownish layer on the dorsal surface of the tongue.

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This layer is primarily composed of desquamated epithelial cells, food debris, leukocytes, mucins, and a significant number of microorganisms, especially anaerobic bacteria. Often underestimated, lingual coating can indicate poor oral hygiene, systemic disorders, or imbalances in the oral microbiome.

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✅ Clinical Characteristics of Lingual Coating
The main clinical features include:

° Whitish or yellowish appearance on the dorsal surface of the tongue.
° Pastelike or sticky texture.
° Halitosis (bad breath) present in most cases.
° Dry mouth sensation or unpleasant taste.
° Possible dysgeusia (altered taste) or burning tongue sensation.

The most commonly affected area is the posterior dorsal region, where filiform papillae are more abundant, facilitating retention of debris and microorganisms.

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✅ Microbiota Involved in Lingual Coating
The tongue harbors a complex microbiota of both aerobic and anaerobic bacteria. Lingual coating is especially associated with a high concentration of Gram-negative anaerobic bacteria that produce volatile sulfur compounds (VSCs), the main cause of halitosis.

➤ Key bacterial species identified:
° Porphyromonas gingivalis
° Fusobacterium nucleatum
° Prevotella intermedia
° Treponema denticola
° Tannerella forsythia
° Solobacterium moorei

These bacteria break down proteins from food and epithelial cells, producing VSCs such as hydrogen sulfide (H₂S), methyl mercaptan, and dimethyl sulfide, which are highly volatile and malodorous.
A recent metagenomic study by Seerangaiyan et al. (2017) confirmed that the tongue is one of the most microbiologically diverse sites in the oral cavity.

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✅ Causes of Lingual Coating
Several factors contribute to the development of lingual coating:

1. Poor oral hygiene
Failure to brush the tongue promotes debris and microbial buildup.
2. Xerostomia (dry mouth)
Reduced salivary flow impairs the self-cleansing ability of the tongue.
3. Tobacco and alcohol use
These habits disrupt the oral flora and irritate the tongue’s surface.
4. Systemic diseases
Conditions like diabetes, chronic kidney disease, or gastrointestinal infections can manifest with tongue coating.
5. Use of antibiotics or antiseptic mouthwashes
Certain medications can alter the oral microbiome.

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✅ Clinical Consequences of Lingual Coating
Though often considered minor, lingual coating can lead to significant consequences:

° Chronic halitosis: The primary source of intraoral bad breath (Morita & Wang, 2001).
° Taste alterations: Due to mechanical obstruction of taste buds.
° Psychological discomfort: Bad breath may lead to social anxiety or isolation.
° Risk of periodontal disease: Lingual bacteria can colonize the gums.
° Indicator of systemic conditions: May be an early sign of candidiasis or immunosuppression.

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✅ Prevention of Lingual Coating
Effective preventive strategies include:

° Daily tongue cleaning: Using tongue scrapers or brushes with a cleaner surface.
° Proper oral hygiene: Brushing and flossing to reduce overall biofilm.
° Antimicrobial mouth rinses: Chlorhexidine 0.12% or zinc lactate in severe cases.
° Adequate hydration: To stimulate saliva production.
° Avoidance of tobacco and alcohol.
° Regular dental visits: For professional evaluation and monitoring.

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✅ Treatment of Lingual Coating
Treatment should be multifactorial and individualized, addressing the underlying cause:

1. Tongue scraping
° Mechanical removal of coating using specialized tools—highly effective and affordable.
2. Antimicrobial therapy
° Rinses with chlorhexidine, cetylpyridinium chloride, or chlorine dioxide to reduce microbial load.
° Antibacterial toothpastes may support overall reduction of pathogens.
3. Correction of predisposing factors
° Manage dry mouth and systemic conditions (e.g., diabetes, kidney failure).
° Eliminate irritants such as smoking or alcohol.
4. Complementary therapies
° Oral probiotics (e.g., Lactobacillus reuteri) to modulate microbiota.
° Patient education on hygiene techniques and healthy lifestyle choices.

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💡 Conclusion
Lingual coating is more than an aesthetic concern. It reflects microbial imbalances in the oral ecosystem and can impact systemic and psychosocial health. Its prevention and treatment are simple yet require consistency and patient education. A comprehensive approach—addressing both local and systemic factors—is essential for effective management.

📚 References

✔ Morita, M., & Wang, H. L. (2001). Association between oral malodor and adult periodontitis: a review. Journal of Clinical Periodontology, 28(9), 813–819. https://doi.org/10.1034/j.1600-051x.2001.028009813.x

✔ Seerangaiyan, K., Jüch, F., Winkel, E. G., & Winkelhoff, A. J. V. D. (2017). Tongue Microbiome in Healthy Subjects and Patients with Intra-Oral Halitosis. Journal of Breath Research, 11(3), 036010. https://doi.org/10.1088/1752-7163/aa6f9e

✔ Takeshita, T., Suzuki, N., Nakano, Y., & Yamashita, Y. (2008). Relationship between oral malodor and the global composition of indigenous bacterial populations in saliva. Applied and Environmental Microbiology, 74(2), 562–569. https://doi.org/10.1128/AEM.02039-07

✔ Tangerman, A., & Winkel, E. G. (2010). Intra- and extra-oral halitosis: findings of a new classification. Journal of Clinical Periodontology, 37(9), 807–814. https://doi.org/10.1111/j.1600-051X.2010.01699.x

✔ Faveri, M., Gonçalves, L. F., Feres, M., Figueiredo, L. C., & de Figueiredo, C. A. (2006). Prevalence and microbiological diversity of tongue coating in subjects with and without halitosis. Revista da Associação Paulista de Cirurgiões Dentistas, 60(2), 150–155.

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