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lunes, 10 de noviembre de 2025

Early vs Delayed Tooth Eruption in Children: Causes, Treatments, and Developmental Implications

Tooth eruption

The timing of tooth eruption is a key indicator of a child’s growth and oral development. Early (premature) or delayed tooth eruption may signal local or systemic conditions. Understanding these variations helps in accurate diagnosis and appropriate treatment.

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Introduction
Tooth eruption is a physiological process involving the movement of teeth from their developmental position in the jaw to their functional position in the oral cavity. Normally, primary teeth erupt between 6 months and 3 years, while permanent teeth appear between 6 and 13 years.
However, when eruption occurs significantly earlier or later than these expected ranges, it can indicate nutritional deficiencies, hormonal imbalances, genetic syndromes, or local obstructions such as cysts or supernumerary teeth (Kumar et al., 2022).

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1. Early Tooth Eruption (Precocious Eruption)
Early eruption can occur in natal or neonatal teeth, or in cases where permanent teeth appear before expected age.

Common causes include:
▪️ Genetic predisposition.
▪️ Endocrine disorders such as hyperthyroidism.
▪️ Local factors like premature loss of primary teeth.

Clinically, early eruption may cause feeding difficulties, gingival irritation, and an increased risk of dental caries due to immature enamel structure.

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2. Delayed Tooth Eruption
Delayed eruption is defined as tooth emergence occurring six months or more beyond the expected time for a given tooth.

Possible causes include:
▪️ Nutritional deficiencies (vitamin D, calcium).
▪️ Endocrine disorders (hypothyroidism, hypopituitarism).
▪️ Genetic syndromes (Down syndrome, cleidocranial dysplasia).
▪️ Local factors, including cysts, trauma, or crowding.

Radiographic evaluation helps rule out obstruction or impaction. Treatment depends on addressing the underlying cause—ranging from nutritional supplementation to orthodontic intervention.

📊 Average Eruption Ages of Primary and Permanent Teeth

Tooth Type Average Eruption Age (Primary Dentition) Average Eruption Age (Permanent Dentition)
Central Incisor 6–10 months (lower), 8–12 months (upper) 6–8 years
Lateral Incisor 9–13 months 7–9 years
Canine 16–22 months 9–12 years
First Molar 12–18 months 6–7 years
Second Molar 20–30 months 11–13 years
💬 Discussion
Both early and delayed eruption affect occlusion, aesthetics, and oral function. Early eruption can increase caries susceptibility, while delayed eruption may interfere with normal alignment and jaw growth. Pediatric dentists should evaluate eruption chronology charts, medical history, and radiographs before determining treatment. Early identification allows for preventive and interceptive approaches, such as fluoride application, habit correction, or surgical exposure when indicated.

✍️ Conclusion
The timing of tooth eruption varies among children but remains a critical diagnostic marker of general health. Clinicians must monitor deviations from eruption norms to prevent complications in occlusion and function. Regular dental check-ups from early childhood are essential to detect eruption anomalies promptly.

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🔎 Recommendations
▪️ Maintain periodic dental evaluations from the first year of life.
▪️ Use eruption charts as reference tools for growth assessment.
▪️ Investigate any eruption delay exceeding six months.
▪️ Coordinate with pediatricians to address systemic causes.
▪️ Encourage balanced nutrition and oral hygiene to promote healthy eruption.

📚 References

✔ Kumar, A., Gupta, R., & Sharma, S. (2022). Assessment of eruption timing and sequence in Indian children: A cross-sectional study. Journal of Indian Society of Pedodontics and Preventive Dentistry, 40(3), 245–250. https://doi.org/10.4103/JISPPD.JISPPD_199_21
✔ Seow, W. K. (2018). Eruption disturbances of the primary and permanent dentitions in children. Australian Dental Journal, 63(S1), S55–S65. https://doi.org/10.1111/adj.12591
✔ Moslemi, M. (2021). An epidemiologic survey of the time and sequence of eruption of permanent teeth in 4–15-year-olds. Journal of Dentistry (Tehran), 18(4), 251–259.

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domingo, 9 de noviembre de 2025

Local Anesthesia Techniques in Children: Effectiveness and Comfort in Pediatric Dentistry

Dental Anesthesia

Local anesthesia in pediatric dentistry is essential for pain control, cooperation, and treatment success. This article reviews the most effective anesthesia techniques, evaluates comfort levels in children, and discusses common complications and preventive strategies.

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Introduction
Pain management in pediatric dentistry is crucial for promoting positive dental experiences and reducing future anxiety. The administration of local anesthesia (LA) allows clinicians to perform invasive procedures while maintaining comfort and cooperation. However, children differ anatomically and psychologically from adults, requiring adapted injection techniques and behavioral strategies.

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The goal of this article is to analyze the effectiveness and comfort of local anesthesia techniques used in children and their impact on clinical success and patient behavior.

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Main Techniques of Local Anesthesia in Pediatric Dentistry

1. Infiltration Anesthesia
This is the most commonly used technique in pediatric patients due to the porosity of the maxillary bone, which allows easy diffusion of anesthetic solutions.
▪️ Indicated for: Extractions, restorations, and minor surgical procedures in the maxilla.
▪️ Advantages: High success rate, minimal discomfort, easy application.
▪️ Limitations: Limited effect on mandibular molars due to dense cortical bone.

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2. Inferior Alveolar Nerve Block
Used for mandibular procedures, this technique requires anatomical precision.
▪️ Advantages: Effective for multiple mandibular teeth.
▪️ Limitations: Higher risk of complications such as hematoma, trismus, or nerve injury, and more discomfort during administration.
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3. Intraligamentary and Intrapulpal Anesthesia
Applied when traditional techniques are insufficient or contraindicated.
▪️ Advantages: Immediate onset, minimal systemic absorption.
▪️ Limitations: May cause postoperative discomfort or localized tissue damage.

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4. Topical and Computer-Controlled Local Anesthesia (CCLAD)
Topical anesthetics reduce the pain of needle insertion, while computer-assisted delivery systems control injection pressure and flow.
▪️ Advantages: Increased comfort and reduced anxiety.
▪️ Limitations: Requires specialized equipment and cost considerations.

💬 Discussion
Pain perception in children depends on age, fear level, and prior dental experience. Studies show that techniques like infiltration and CCLAD produce higher comfort and cooperation levels compared to nerve blocks (Ram & Peretz, 2021).
The choice of anesthetic solution (e.g., lidocaine, articaine, mepivacaine) influences onset time and duration. Articaine, for instance, offers faster onset and better diffusion, making it suitable for infiltration in both arches (Kämmerer et al., 2020).
Behavioral management techniques, including tell-show-do, distraction, and parental reassurance, complement anesthesia and reduce fear-related resistance.

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✍️ Conclusion
Effective local anesthesia in children ensures pain-free and cooperative dental treatment, contributing to a lifetime of positive dental attitudes. Technique selection should depend on the child’s anatomy, age, and anxiety level. Incorporating technological advances like CCLAD systems and topical anesthetics enhances comfort and minimizes complications.

🔎 Recommendations
▪️ Evaluate each child’s medical and behavioral profile before choosing the technique.
▪️ Use topical anesthesia and gentle injection techniques to minimize discomfort.
▪️ Prefer infiltration anesthesia for most pediatric cases; use nerve blocks only when necessary.
▪️ Train clinicians in behavioral management and pain-control technologies.
▪️ Implement post-anesthesia follow-up to detect and manage complications early.

📊 Common Complications of Local Anesthesia in Children

Aspect Advantages Limitations
Soft tissue injury (lip or cheek biting) Usually self-limiting; minor tissue damage Pain, swelling, and ulceration post-anesthesia
Hematoma Rare; resolves spontaneously Facial swelling and discomfort; aesthetic concern
Trismus Indicates tissue reaction and can guide technique improvement Difficulty in opening mouth; may affect eating or speech
Nerve injury Extremely rare; transient in most cases May cause temporary paresthesia or altered sensation
Allergic reaction Identifies sensitivity for future anesthetic selection Potentially severe; requires emergency management

📚 References

✔ Kämmerer, P. W., Schneider, D., & Daubländer, M. (2020). Clinical comparison of 4% articaine with 1:100,000 epinephrine versus 2% lidocaine with 1:100,000 epinephrine for local infiltration anesthesia in children. Clinical Oral Investigations, 24(1), 411–417.
✔ Ram, D., & Peretz, B. (2021). Efficacy of computer-controlled local anesthetic delivery system in pediatric dental patients. Journal of Clinical Pediatric Dentistry, 45(2), 82–87.
✔ Sarmiento, S., & Ramírez, A. (2022). Complications of local anesthesia in pediatric dental patients: A review. International Journal of Paediatric Dentistry, 32(3), 345–352.
✔ Malamed, S. F. (2019). Handbook of Local Anesthesia (7th ed.). Elsevier.

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Differential Diagnosis of White Lesions in the Pediatric Oral Mucosa

White Lesions

White lesions in the pediatric oral cavity represent a diagnostic challenge due to their diverse etiology. Accurate diagnosis is essential to distinguish benign, infectious, and potentially malignant conditions.

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This article reviews the main causes, diagnostic criteria, and management strategies for white lesions in the oral mucosa of children.

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Introduction
White lesions in the oral mucosa are frequent findings in pediatric dental practice. They may range from simple keratotic changes to fungal infections or early mucosal diseases. A systematic clinical approach is crucial to prevent misdiagnosis and inappropriate treatment.

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1. Common Causes of White Lesions in Children
The most prevalent conditions include:

▪️ Frictional keratosis due to chronic trauma from dental appliances or biting.
▪️ Candidiasis, commonly caused by Candida albicans, appearing as removable white plaques.
▪️ Leukoplakia, a rare but important potentially malignant lesion.
▪️ Lichen planus, uncommon in children but characterized by reticular white striations.
▪️ Chemical burns from topical medicaments or toothpastes.
▪️ Congenital disorders such as White Sponge Nevus.

Each condition presents distinct clinical features, necessitating thorough anamnesis and visual-tactile examination.

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2. Diagnostic Considerations
Diagnosis should begin with:

▪️ Detailed clinical evaluation (location, symmetry, texture, and presence of pain).
▪️ History taking, including trauma, medications, or recent infections.
▪️ Removal test, as in candidiasis where the lesion can be wiped away.
▪️ Biopsy and cytology, recommended for persistent or atypical lesions.

Use of adjunctive diagnostic tools, such as toluidine blue staining or autofluorescence, may aid in differentiating potentially malignant conditions.

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💬 Discussion
The differential diagnosis of pediatric white lesions requires distinguishing between reversible inflammatory lesions and potentially precancerous alterations. While traumatic and fungal lesions are the most common, clinicians should remain alert for unusual presentations such as leukoplakia or lichen planus.
Early diagnosis allows for conservative treatment and prevention of complications. Overdiagnosis, however, should be avoided through confirmatory tests and follow-up evaluations.

✍️ Conclusion
White lesions in the pediatric oral mucosa are mostly benign and transient, but some may indicate systemic or precancerous conditions. Clinical observation, microbiological analysis, and biopsy when indicated are key to correct diagnosis and management. Dental professionals should maintain a structured diagnostic protocol to ensure patient safety and accurate intervention.

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🔎 Recommendations
▪️ Conduct a comprehensive oral examination in all pediatric patients.
▪️ Educate parents about avoiding chemical irritants and oral trauma.
▪️ Treat fungal infections early and reassess lesions that persist beyond 2 weeks.
▪️ Refer for biopsy any lesion with induration, ulceration, or unexplained persistence.

📊 Summary Table: Differential Diagnosis of Pediatric Oral White Lesions

Aspect Advantages Limitations
Clinical observation Non-invasive, allows identification of trauma or fungal infection May not differentiate early neoplastic changes
Microbiological tests Useful for diagnosing candidiasis and bacterial superinfections Limited utility for keratotic or autoimmune lesions
Biopsy and histopathology Definitive diagnosis for atypical or persistent lesions Invasive, requires child cooperation and healing period
Follow-up examination Allows monitoring of lesion evolution and treatment response Requires long-term compliance from caregivers

📚 References

✔ Al-Maweri, S. A., Al-Soneidar, W. A., & Halboub, E. (2020). Oral mucosal lesions in children: A systematic review. Dental Research Journal, 17(6), 401–409.
✔ Farah, C. S., & McCullough, M. J. (2019). Oral white lesions: An updated clinical diagnostic approach. Journal of Dental Research, 98(7), 715–728.
✔ Mortazavi, H., Baharvand, M., & Mehdipour, M. (2021). Oral white lesions: An updated clinical diagnostic decision tree. Dental Research Journal, 18(1), 22.
✔ Glick, M. (Ed.). (2021). Burket’s Oral Medicine (13th ed.). Wiley-Blackwell.

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Causes and Complications of Dental Implant Failure

Dental Implant Failure

Dental implantology has revolutionized oral rehabilitation, offering high success rates exceeding 95%. However, implant failure remains a critical challenge influenced by biological, mechanical, and procedural factors.

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This article explores the main causes, complications, and preventive measures to ensure predictable outcomes in dental implant therapy.

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Introduction
A dental implant is a biocompatible titanium or zirconium fixture surgically placed into the jawbone to support a prosthetic tooth. Its success depends on osseointegration, a direct structural and functional connection between bone and the implant surface. Despite technological advances, implant failures still occur due to multiple factors that can be biological (infection, bone loss) or mechanical (fracture, overload).
Understanding these causes and implementing proper pre- and postoperative care is essential for minimizing risk and improving patient outcomes.

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1. Etiology of Dental Implant Failure
Dental implant failure can be early (before osseointegration) or late (after loading).

▪️ Early failures result from surgical trauma, infection, or poor bone quality.
▪️ Late failures are often related to peri-implantitis, occlusal overload, or prosthetic complications.

➤ Common causes include:
▪️ Infection and poor asepsis during surgery
▪️ Inadequate primary stability
▪️ Overheating of bone during drilling (>47°C causes necrosis)
▪️ Systemic conditions such as diabetes or smoking
▪️ Insufficient bone volume or density
▪️ Improper prosthetic loading

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2. Biological and Mechanical Complications

➤ Biological complications include:
▪️ Peri-implant mucositis: inflammation limited to soft tissues
▪️ Peri-implantitis: infection leading to bone loss and implant mobility
▪️ Failure of osseointegration

➤ Mechanical complications include:
▪️ Fracture of screws or abutments
▪️ Prosthetic loosening
▪️ Implant fracture due to overload

These complications can lead to loss of the implant and compromised esthetic and functional outcomes.

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3. Risk Factors

▪️ Poor oral hygiene and plaque accumulation
▪️ Smoking and alcohol use
▪️ Uncontrolled systemic diseases (e.g., diabetes mellitus)
▪️ Bruxism and occlusal stress
▪️ Lack of proper surgical planning or experience

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4. Prevention and Management
Prevention begins with comprehensive patient evaluation, including medical history, imaging (CBCT), and treatment planning. Management requires early detection and intervention, such as:

▪️ Antiseptic rinses (chlorhexidine 0.12–0.2%)
▪️ Antibiotic therapy if infection develops
▪️ Regular professional maintenance
▪️ Occlusal adjustment to minimize overload

📊 Comparative Table: Pre- and Post-Surgical Care for Dental Implant Placement

Aspect Pre-Surgical Care Post-Surgical Care
Patient Evaluation Medical history, CBCT imaging, bone quality assessment Monitor healing, control systemic conditions
Surgical Preparation Antiseptic rinse, sterile environment, adequate anesthesia Antibiotic prophylaxis, pain control, antiseptic rinses
Surgical Technique Use irrigation to avoid overheating, ensure primary stability Avoid trauma, assess osseointegration before loading
Oral Hygiene Educate patient on hygiene before procedure Soft brushing, avoid manipulation of the surgical site
Follow-up Schedule visits for implant integration evaluation Periodic check-ups for maintenance and tissue health
💬 Discussion
Despite advances in implant surface technology and guided surgery, implant failure remains multifactorial. Biological integration can be compromised by host factors, surgical technique, and postoperative management. Clinicians must adopt evidence-based protocols for site preparation, asepsis, and maintenance to reduce complications.
Additionally, interdisciplinary collaboration between oral surgeons, prosthodontists, and hygienists improves treatment predictability.

🔎 Recommendations
▪️ Conduct comprehensive medical and radiographic assessments before surgery.
▪️ Use aseptic techniques and ensure adequate irrigation to prevent bone overheating.
▪️ Educate patients on oral hygiene and maintenance protocols.
▪️ Avoid immediate loading in poor bone quality sites.
▪️ Schedule regular follow-ups to monitor tissue health and implant stability.

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✍️ Conclusion
Dental implant failure results from a combination of biological, mechanical, and procedural factors. Effective prevention relies on meticulous case selection, surgical technique, and postoperative care. Understanding these causes and complications enables clinicians to maintain high long-term success rates and patient satisfaction.

📚 References

✔ Esposito, M., Grusovin, M. G., & Worthington, H. V. (2012). Interventions for replacing missing teeth: Treatment of peri-implantitis. Cochrane Database of Systematic Reviews, (12), CD004970. https://doi.org/10.1002/14651858.CD004970.pub5
✔ Pjetursson, B. E., & Heimisdottir, K. (2018). Dental implants – are they better than natural teeth?. European Journal of Oral Sciences, 126(S1), 81–87. https://doi.org/10.1111/eos.12543
✔ Kwon, T., Bain, P. A., Levin, L. (2019). Systemic diseases and implant failure: A review of the literature. International Journal of Oral & Maxillofacial Implants, 34(1), 45–54. https://doi.org/10.11607/jomi.6772

viernes, 7 de noviembre de 2025

How Do Sodas Affect Your Teeth? Understanding the Hidden Dental Risks

Dental Erosion

Sodas are among the most consumed beverages worldwide, but their high acidity and sugar content make them harmful to dental health.

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This article explores how sodas — both regular and diet — affect teeth, their clinical implications, and effective preventive strategies.

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Introduction
Sodas are carbonated beverages containing phosphoric acid, citric acid, and sugar substitutes, which can soften and demineralize tooth enamel. Despite being popular, their regular intake is strongly associated with dental erosion, caries, and hypersensitivity. Understanding these effects is crucial for prevention and patient education in dental practice.

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1. Composition and pH Levels
Regular sodas contain high concentrations of sucrose or fructose and have a pH between 2.5 and 3.5, which significantly reduces the oral environment’s capacity to neutralize acids. Diet sodas, though sugar-free, often include citric acid and carbonic acid, which still contribute to enamel demineralization.

2. Mechanism of Damage
The acidic nature of sodas leads to erosion of the enamel surface, the first stage of structural tooth loss. Repeated exposure causes loss of enamel hardness and exposes dentin, leading to sensitivity and higher susceptibility to decay.

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3. Clinical Manifestations
Patients who frequently consume sodas may present:
▪️ Smooth, shiny enamel surfaces indicating erosion
▪️ Increased tooth sensitivity, especially to cold
▪️ Cervical lesions or cupping of enamel
▪️ Staining and discoloration due to enamel thinning

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4. Risk Factors
▪️ High consumption frequency (especially sipping over time)
▪️ Poor oral hygiene
▪️ Reduced salivary flow
▪️ Concurrent intake of acidic foods (e.g., citrus or vinegar)

📊 Comparative Table: Regular vs. Diet Sodas

Aspect Regular Soda Diet Soda
Main Components Sugar, phosphoric acid, caffeine, carbonation Artificial sweeteners, citric acid, carbonation
pH Level 2.5 – 3.5 (highly acidic) 3.0 – 3.6 (acidic)
Dental Impact Enamel erosion, caries, discoloration Enamel erosion, no direct caries due to lack of sugar
Advantages None for oral health; provides energy (temporary) Low-calorie alternative; reduces sugar intake
Limitations Causes enamel erosion and cavities Still causes enamel erosion due to acidity
💬 Discussion
Although diet sodas eliminate sugars, they remain acidic enough to damage enamel. Thus, switching to diet versions does not completely eliminate risk. The pattern of consumption (slow drinking, bedtime intake, or no rinsing after) exacerbates the harmful effects.
Clinical management should focus on education, fluoride application, remineralizing agents, and reducing consumption frequency rather than complete restriction.

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🔎 Recommendations
▪️ Limit soda intake and prefer water or milk.
▪️ Avoid brushing immediately after consuming acidic beverages; wait at least 30 minutes.
▪️ Use fluoride toothpaste and rinse with neutralizing mouthwash.
▪️ Promote sugar-free chewing gum to stimulate salivation.
▪️ Encourage regular dental check-ups for early detection of erosion.

✍️ Conclusion
Sodas cause progressive damage to dental enamel through acid exposure and demineralization. Both regular and diet sodas can lead to erosion, sensitivity, and increased caries risk. Preventive education, fluoride therapy, and dietary counseling are key to reducing their long-term effects.

📚 References

✔ Hu, D., Li, Y., & Dong, Q. (2020). Effects of acidic beverages on enamel erosion and remineralization: A review. Frontiers in Nutrition, 7, 585856. https://doi.org/10.3389/fnut.2020.585856
✔ Lussi, A., & Carvalho, T. S. (2015). Erosive tooth wear: A multifactorial condition of growing concern and increasing knowledge. Monographs in Oral Science, 25, 1–15. https://doi.org/10.1159/000360380
✔ Zero, D. T. (2016). Etiology of dental erosion—Extrinsic factors. European Journal of Oral Sciences, 124(2), 119–135. https://doi.org/10.1111/eos.12262

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