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martes, 11 de agosto de 2026

Soft Tissue Injuries in Pediatric Dentistry: Assessment and Care

Soft Tissue Injuries

Soft tissue injuries in pediatric dentistry are common consequences of falls, sports-related trauma, collisions, and accidental biting.

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They may involve the lips, gingiva, buccal mucosa, tongue, frenula, and oral vestibule, and can occur with or without associated dental or facial injuries.

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Epidemiological data indicate that soft tissue trauma is particularly frequent in children younger than 3 years.
Although many oral soft tissue injuries heal without intervention, careful assessment is essential to identify deep lacerations, foreign bodies, neurovascular injury, significant hemorrhage, tissue avulsion, mandibular trauma, and injuries requiring surgical repair.
The clinician should also consider the possibility of non-accidental injury when the history and clinical findings are inconsistent.

Types of Pediatric Oral Soft Tissue Injuries
Common injuries include:

▪️ Lacerations: Partial- or full-thickness tissue tears affecting the lips, tongue, gingiva, or buccal mucosa.
▪️ Contusions: Blunt trauma producing localized swelling, bruising, or tenderness without tissue disruption.
▪️ Abrasions: Superficial epithelial injuries caused by friction.
▪️ Puncture wounds: Penetrating injuries that may retain foreign material.
▪️ Frenulum injuries: Often associated with falls or direct impact to the upper lip.
▪️ Gingival degloving injuries: Separation of the gingiva from the underlying alveolar bone, which may be clinically subtle.
▪️ Tongue lacerations: Frequently caused by falls or accidental biting.
The injury should always be interpreted in the context of the mechanism of trauma and the possibility of concomitant dental, alveolar, mandibular, or head injuries.

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Assessment of Soft Tissue Injuries

1. Establish the Mechanism and Timing
The history should document the mechanism of injury, time elapsed, location, witnessed circumstances, initial bleeding, previous first aid, medical history, medications, allergies, and tetanus immunization status. The clinician should determine whether the trauma was accidental and whether the reported mechanism adequately explains the observed injuries.

2. Perform a Systematic Examination
A complete examination should include:

▪️ Extraoral inspection for swelling, bruising, asymmetry, and lacerations.
▪️ Intraoral inspection of the lips, gingiva, mucosa, tongue, floor of the mouth, and palate.
▪️ Assessment of wound depth, tissue separation, contamination, and foreign bodies.
▪️ Evaluation of hemostasis and tissue perfusion.
▪️ Assessment of occlusion and mandibular movement.
▪️ Examination of teeth for fracture, mobility, displacement, or avulsion.
▪️ Evaluation for neurological symptoms or associated facial injuries.
The lips should be carefully retracted to identify injuries hidden within the vestibule or gingival tissues. Missing teeth or fragments should be accounted for because they may be embedded in soft tissue or, less commonly, aspirated.

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When Does a Soft Tissue Injury Require Repair?
Most minor oral lacerations can heal by secondary intention because the oral mucosa has a favorable blood supply and healing capacity. Suturing should therefore not be automatic.

Repair or specialist evaluation should be considered when there is:
▪️ Persistent or uncontrolled bleeding.
▪️ Significant tissue separation or a large flap.
▪️ Deep or through-and-through laceration.
▪️ Injury involving important anatomical structures.
▪️ Significant contamination or retained foreign bodies.
▪️ Tissue avulsion or compromised vascularity.
▪️ Functional impairment.
▪️ Facial laceration requiring precise anatomical alignment.
▪️ Suspected associated fracture or penetrating injury.
Deep oral lacerations and gingival degloving injuries may require operative irrigation, debridement, and closure to reduce complications.

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Tongue Lacerations in Children
Tongue injuries deserve particular consideration because clinicians may overestimate the need for suturing. Evidence indicates that many uncomplicated tongue lacerations heal satisfactorily without primary closure.
In a retrospective pediatric study, suturing was generally unnecessary for gaping tongue lacerations smaller than 2 cm that did not involve the tongue tip. Larger wounds, wounds with significant gaping at rest, through-and-through injuries, or injuries involving the tip may require repair depending on their anatomical and functional characteristics.
Therefore, wound size alone should not determine treatment. Location, depth, tissue loss, bleeding, functional impairment, and wound configuration should guide the decision.

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Management and Wound Care
Initial treatment consists of:

1. Hemostasis using direct pressure when appropriate.
2. Irrigation and removal of visible foreign material.
3. Assessment for associated dental and maxillofacial injuries.
4. Adequate local anesthesia and analgesia before wound manipulation.
5. Primary closure when clinically indicated.
6. Appropriate postoperative instructions and follow-up.
In children requiring extensive examination or repair, behavioral cooperation may be insufficient for safe treatment under local anesthesia alone. Depending on the injury and clinical setting, sedation or general anesthesia may be necessary.
Routine systemic antibiotics are not indicated for every uncomplicated oral laceration. Their use should be based on wound contamination, tissue damage, operative management, associated injuries, and patient-specific risk factors. Antibiotic prophylaxis may be considered for contaminated soft tissue injuries requiring operative intervention.

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Special Considerations in Pediatric Patients
Children present unique challenges because of their age, developmental stage, limited ability to cooperate, and the potential psychological impact of traumatic procedures. Treatment should therefore combine adequate pain control, efficient wound assessment, appropriate behavior guidance, and avoidance of unnecessary invasive procedures.
The clinician should also recognize that an oral injury in a non-mobile infant, particularly when the reported mechanism is unclear or inconsistent with the clinical findings, warrants careful consideration of possible child maltreatment. Oral injuries associated with physical abuse may include frenulum trauma, bruising, lacerations, and injuries to the oropharyngeal region.

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💬 Discussion
The management of pediatric oral soft tissue injuries is primarily determined by clinical assessment rather than by wound size alone. Contemporary evidence supports a conservative approach for many uncomplicated mucosal injuries, avoiding unnecessary suturing when spontaneous healing is expected to provide an equivalent outcome.
The main clinical challenge is distinguishing injuries appropriate for observation from those requiring intervention. Particular attention should be directed toward deep lacerations, tissue avulsion, persistent hemorrhage, functional impairment, contaminated wounds, gingival degloving, and associated facial or dental trauma.
For tongue lacerations specifically, available evidence suggests that routine suturing may increase treatment burden without improving outcomes in selected uncomplicated wounds. However, the evidence base remains limited, and management should be individualized according to anatomical involvement and clinical severity.

🎯 Clinical Recommendations
▪️ Prioritize airway, neurological status, hemorrhage, and associated facial trauma before focusing exclusively on the oral wound.
▪️ Document the injury with a precise description of location, dimensions, depth, contamination, tissue loss, and associated dental findings.
▪️ Avoid routine suturing of uncomplicated oral mucosal wounds when secondary healing is clinically appropriate.
▪️ Consider repair for wounds with significant gaping, tissue loss, persistent bleeding, functional compromise, or important anatomical involvement.
▪️ For tongue lacerations, evaluate location, depth, gaping at rest, tip involvement, and through-and-through extension rather than relying solely on length.
▪️ Use adequate analgesia and anesthesia before wound exploration or repair; consider sedation when cooperation is inadequate for safe treatment.
▪️ Maintain a low threshold for referral when deep facial/oral structures, mandibular injury, vascular compromise, or complex soft tissue trauma is suspected.
▪️ Reassess the reported mechanism when the injury pattern is inconsistent, particularly in non-mobile infants.

✍️ Conclusion
Soft tissue injuries in pediatric dentistry require a structured clinical assessment to determine whether observation, wound care, primary repair, or specialist referral is appropriate. Most uncomplicated oral mucosal injuries heal effectively without surgical intervention. However, deep lacerations, degloving injuries, significant tissue loss, uncontrolled bleeding, functional impairment, and associated maxillofacial trauma require more intensive management.
A conservative, evidence-informed approach can minimize unnecessary procedures while preserving function, tissue integrity, and the child's overall experience of dental care.

📚 References

✔ American Academy of Pediatric Dentistry. (2025). Acute traumatic injuries: Assessment and documentation. In The reference manual of pediatric dentistry. American Academy of Pediatric Dentistry.
Hwang, M., Engelstad, M., & Chandra, S. R. (2023). Management of soft tissue injuries in children—A comprehensive review. Oral and Maxillofacial Surgery Clinics of North America, 35(4), 619–629. https://doi.org/10.1016/j.coms.2023.06.003
✔ Lamell, C. W., Fraone, G., Casamassimo, P. S., & Wilson, S. (1999). Presenting characteristics and treatment outcomes for tongue lacerations in children. Pediatric Dentistry, 21(1), 34–38.
✔ Olszewska, A., Kensy, J., Czajka-Jakubowska, A., Pergolini, D., Bossù, M., Romeo, U., & Matys, J. (2026). Diagnosis and management of traumatic injuries in pediatric patients secondary to dental local anesthesia: A systematic review. Advances in Clinical and Experimental Medicine, 35(1), 175–190. https://doi.org/10.17219/acem/204391
✔ Seiler, M., Massaro, S. L., Staubli, G., & Schiestl, C. (2018). Tongue lacerations in children: To suture or not? Swiss Medical Weekly, 148, w14683. https://doi.org/10.4414/smw.2018.14683
✔ The Royal Children's Hospital Melbourne. (2024). Clinical practice guidelines: Dental trauma.
✔ The Royal Children's Hospital Melbourne. (2022). Clinical practice guidelines: Lacerations.
✔ Warnakulasuriya, S., Muthukrishnan, A., et al. (2019). World Workshop on Oral Medicine VII: Relative frequency of oral mucosal lesions in children, a scoping review. Oral Diseases, 25(8), 1933–1948. https://doi.org/10.1111/odi.13112

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lunes, 10 de agosto de 2026

Silver Diamine Fluoride for Root Caries: Is It Effective?

Silver Diamine Fluoride

Root caries is an increasing clinical concern in adults and older adults, particularly when gingival recession exposes root surfaces. Unlike enamel, exposed root dentin and cementum are more susceptible to acid-mediated demineralization.

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Management can be challenging when lesions occur in patients with multiple affected surfaces, limited access to dental care, reduced oral hygiene capacity, or medical and functional limitations.

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Silver diamine fluoride (SDF), particularly the 38% formulation, has emerged as a nonrestorative approach for arresting root caries. Its clinical value is based on the ability to inhibit caries progression without requiring conventional cavity preparation.
Current evidence supports SDF as an option for arresting root caries in permanent teeth, although the certainty of evidence and clinical recommendations vary according to the treatment alternative and patient circumstances.

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Why Is Root Caries Difficult to Manage?
Root caries develops on exposed root surfaces after loss of periodontal coverage. Dentin and cementum have a higher organic content and are less mineralized than enamel, making them more vulnerable to demineralization.
Root caries management may be particularly challenging in older adults because lesions can occur in multiple teeth and may be associated with gingival recession, plaque accumulation, xerostomia, reduced manual dexterity, or difficulty maintaining oral hygiene.
Conventional restorative treatment remains appropriate when a lesion requires restoration because of structural loss, esthetic requirements, function, or other clinical considerations. However, a nonrestorative strategy can be useful when the primary objective is to arrest disease progression.

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How Does SDF Work on Root Caries?
The anticaries effects of SDF are related primarily to its silver and fluoride components.
Silver has antimicrobial properties that can inhibit cariogenic microorganisms and interfere with bacterial activity within the carious lesion. Fluoride contributes to the formation and stabilization of fluoride-containing mineral phases and increases the resistance of dental hard tissues to subsequent acid attack.
For root caries, the objective is not to restore the original anatomy of the tooth. Instead, SDF is used to inactivate the carious lesion and prevent or reduce further progression.
Clinically, an arrested lesion generally becomes harder and darker after treatment. The permanent dark discoloration is an important consequence of SDF therapy and should be incorporated into informed consent.

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What Does the Clinical Evidence Show?
Evidence from randomized clinical trials and systematic reviews supports the effectiveness of SDF for root caries in older adults.
A systematic review and meta-analysis evaluating SDF for exposed root surfaces included three controlled clinical trials involving 895 older adults. Compared with placebo, SDF significantly reduced the number of decayed or filled root surfaces at 24 months and at 30 months or longer. SDF also showed preventive effects comparable to chlorhexidine and fluoride varnish in the included studies.
A subsequent systematic review and meta-analysis published in 2021 similarly concluded that SDF was effective for the management of root caries in older adults, while emphasizing the limitations of the available evidence.

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Evidence From a Recent Randomized Clinical Trial
More recent evidence further supports the caries-arresting effect of 38% SDF.
A 2026 randomized controlled trial included 186 community-dwelling older adults, with 323 root caries lesions assessed at baseline. At the 2-month follow-up, 165 participants with 286 lesions remained in the analysis.
The SDF group demonstrated substantially higher arrest rates than the control group:

▪️ Individual level: 81.2% with SDF vs. 21.3% with control
▪️ Lesion level: 84.9% with SDF vs. 20.7% with control
The study also found that SDF application, baseline lesion status, and absence of plaque on the lesion surface were positively associated with caries arrest.
Although the short follow-up limits conclusions regarding long-term effectiveness, these findings provide additional contemporary clinical evidence supporting 38% SDF for arresting root caries.

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Is SDF Recommended for Root Caries?
The current evidence-based recommendations are nuanced.
The American Dental Association (ADA) guideline on nonrestorative caries treatment identifies 38% SDF as a nonrestorative option for noncavitated and cavitated root caries lesions on permanent teeth.
However, the ADA guideline prioritizes 5,000 ppm fluoride toothpaste or gel used at least once daily over other nonrestorative options, including annual 38% SDF, because of the overall evidence assessment, feasibility, and clinical considerations. The recommendation for root-surface treatment is conditional and based on low-certainty evidence.
Therefore, SDF should not be interpreted as the universally preferred treatment for every root caries lesion. Its value is particularly relevant when a nonrestorative, minimally invasive approach is appropriate.

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Advantages of SDF for Root Caries
Several characteristics make SDF clinically attractive for selected patients:

▪️ Nonrestorative application: no conventional cavity preparation is required to arrest the lesion.
▪️ Minimal invasiveness: treatment can be performed without rotary instrumentation.
▪️ Caries-arresting activity: clinical studies demonstrate significant arrest of root caries compared with placebo.
▪️ Potential usefulness in older adults: particularly when multiple root lesions or treatment limitations are present.
▪️ Simple topical application: the procedure requires relatively limited clinical resources.
▪️ Potential public-health value: SDF may facilitate disease control in populations with barriers to conventional restorative treatment.
These advantages should be considered within a comprehensive caries-management strategy rather than as a replacement for oral hygiene measures, fluoride exposure, dietary management, or restorative treatment when clinically indicated.

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Limitations of SDF for Root Caries
The principal limitation is permanent dark discoloration of the treated carious lesion. This can be particularly relevant when lesions are located on visible root surfaces.
SDF also does not restore lost tooth structure. An arrested lesion may remain cavitated, and the patient may still require restorative treatment when structural integrity, plaque control, function, food impaction, or esthetics are concerns.
Another limitation is the heterogeneity of the available evidence, including differences in lesion characteristics, application protocols, follow-up periods, and patient populations. The ADA therefore assigns a conditional recommendation with low certainty for nonrestorative management of root caries using SDF.
Importantly, evidence from root caries studies should not automatically be extrapolated to every form of dental caries or to every clinical situation.

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💬 Discussion
The available evidence indicates that 38% silver diamine fluoride is effective in arresting root caries, particularly in older adults. Both earlier systematic reviews and more recent randomized clinical evidence demonstrate a substantially greater likelihood of lesion arrest with SDF than with placebo or control treatment.
Nevertheless, effectiveness should be interpreted in the context of the treatment objective. SDF is primarily a disease-arresting intervention, not a restorative procedure. It can stabilize a carious root surface but does not reconstruct missing dental tissue.
The 2026 randomized trial also highlights the importance of plaque control. The absence of plaque on the lesion surface was positively associated with caries arrest, reinforcing that SDF should be integrated into broader preventive and behavioral management rather than used as an isolated intervention.
Current guidelines consequently position SDF as one component of nonrestorative root-caries management rather than a universal substitute for high-concentration fluoride toothpaste, preventive care, or restorations when indicated.

🎯 Clinical Recommendations
1. Consider 38% SDF for active root caries in permanent teeth when a nonrestorative approach is clinically appropriate.
2. Prioritize comprehensive caries management, including plaque control, appropriate fluoride exposure, dietary counseling, and management of contributing risk factors.
3. Discuss permanent discoloration before treatment, particularly for lesions on visible root surfaces, and obtain appropriate informed consent.
4. Do not regard SDF as a restorative substitute when substantial structural rehabilitation, function, esthetics, or plaque-retentive cavitation requires restoration.
5. Monitor treated lesions clinically for changes in hardness, activity, plaque accumulation, and progression.
6. Use patient-specific decision-making. The ADA currently gives a conditional recommendation with low-certainty evidence for SDF on root surfaces, while prioritizing daily 5,000 ppm fluoride toothpaste or gel as the preferred nonrestorative strategy when feasible.


✍️ Conclusion
Silver diamine fluoride, particularly 38% SDF, has demonstrated clinically significant caries-arresting activity on exposed root surfaces. Evidence from systematic reviews and a recent randomized clinical trial supports its use as a nonrestorative option, especially in older adults and patients for whom conventional restorative treatment may be difficult or undesirable.
However, SDF does not restore lost tooth structure and produces permanent dark staining of treated carious lesions. Its use should therefore be based on lesion characteristics, patient preferences, caries risk, plaque control, functional and esthetic considerations, and the feasibility of alternative preventive or restorative treatments.

📚 References

✔ American Dental Association. (2018). Evidence-based clinical practice guideline on nonrestorative treatments for carious lesions: A report from the American Dental Association. Journal of the American Dental Association, 149(10), 837–849.e19. https://doi.org/10.1016/j.adaj.2018.07.002
✔ Fontana, M., & Weyant, R. J. (2025). Silver diamine fluoride for caries management: An executive summary of the clinical practice guideline. Journal of the California Dental Association, 53(1), Article 2588948. https://doi.org/10.1080/19424396.2025.2588948
✔ Grandjean, M.-L., Maccarone, N. R., McKenna, G., Müller, F., & Srinivasan, M. (2021). Silver diamine fluoride (SDF) in the management of root caries in elders: A systematic review and meta-analysis. Swiss Dental Journal, 131(5), 417–424. https://doi.org/10.61872/sdj-2021-05-02
✔ Zhang, W., McGrath, C., Lo, E. C. M., & Li, J. Y. (2016). Silver diamine fluoride: A systematic review of clinical trials. Journal of Dental Research, 95(5), 512–519. https://doi.org/10.1177/0022034516630671

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sábado, 8 de agosto de 2026

Can Testosterone Affect Orthodontic Tooth Movement?

Orthodontic Tooth Movement

A 2026 animal study published in the Journal of Periodontology investigated how altered testosterone levels affect orthodontic tooth movement (OTM), alveolar bone remodeling, periodontal tissues, and root resorption.

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The findings indicate that both testosterone deficiency and high-dose anabolic-androgenic steroid exposure can modify the biological response to orthodontic force in a pubertal male rat model.

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The study provides experimental evidence that testosterone-related disturbances may influence the tissues involved in orthodontic tooth movement. However, because the research was conducted in rats, the findings cannot be directly applied to human orthodontic treatment.

🔹 Study Design
The researchers used a pubertal male rat model to investigate the effects of testosterone dysfunction during orthodontic tooth movement.
Testosterone deficiency was induced through orchiectomy. The researchers subsequently administered testosterone undecanoate, an anabolic-androgenic steroid (AAS), at replacement and high doses.
Orthodontic tooth movement was produced by applying a closed-coil spring to the maxillary right first molar. The surrounding alveolar bone and periodontal ligament were evaluated at 5 and 10 days after orthodontic force application.
The investigators used several analytical methods, including micro-computed tomography (micro-CT), reverse-transcription quantitative polymerase chain reaction (RT-qPCR), and immunohistochemistry. Root resorption and plasma concentrations of testosterone and adrenocorticotropic hormone were also evaluated.

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🔹 Testosterone Dysfunction Altered Bone Microarchitecture
Both testosterone deficiency and high-dose AAS exposure produced significant changes in bone microarchitecture.
The researchers reported reductions in trabecular thickness and bone connectivity, together with changes involving bone lacunae.
These findings indicate that altered testosterone conditions affected the alveolar bone response during the experimental orthodontic procedure.
The study also found that testosterone dysfunction was associated with greater rotation and intrusion of the orthodontically moved tooth. These observations were obtained from the animal model and should not be interpreted as evidence that testosterone manipulation produces predictable changes in the rate or direction of orthodontic tooth movement in humans.

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🔹 High-Dose Anabolic Steroids and Root Resorption
One of the clinically relevant findings involved inflammatory changes and root resorption.
According to the study, high-dose AAS exposure intensified the inflammatory infiltrate and root resorption associated with orthodontic tooth movement.
Root resorption is a biological consequence that can occur during orthodontic tooth movement. In this experimental model, the researchers found that high-dose androgen exposure was associated with a greater resorptive response.
The study therefore identifies a potential relationship between androgen disturbance and orthodontically induced tissue changes. However, it does not establish that anabolic-androgenic steroid use causes clinically significant root resorption in human orthodontic patients.

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🔹 Changes in Bone-Related Gene Expression
The investigators also examined molecular markers associated with bone metabolism.
Testosterone dysfunction altered the expression of several genes, including:
▪️ Runx2
▪️ Bmp2
▪️ Spp1
▪️ Bglap
The study also identified deregulation of the RANK/RANKL/OPG pathway following testosterone disturbances.
These molecular findings provide additional evidence that altered testosterone conditions affected biological pathways involved in bone remodeling during orthodontic tooth movement.

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🔹 Testosterone Replacement Did Not Fully Restore the Response
An important finding was that administration of AAS at replacement doses did not normalize the inflammatory infiltrate, orthodontic tooth movement, or the expression of the studied genes to control levels.
Therefore, the experimental response observed in testosterone-deficient animals was not simply reversed by testosterone administration at the replacement dose used in the study.
This finding suggests that the biological effects associated with testosterone disturbance were not completely restored under the experimental conditions evaluated.

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🔹 What Do These Findings Mean for Orthodontics?
The study provides evidence that testosterone dysfunction can influence several biological processes associated with orthodontic tooth movement, including:
▪️ alveolar bone microarchitecture;
▪️ periodontal inflammatory response;
▪️ tooth movement characteristics;
▪️ root resorption; and
▪️ expression of genes involved in bone metabolism.
However, the study does not establish a clinical protocol for modifying testosterone levels during orthodontic treatment.
It also does not demonstrate that testosterone supplementation can accelerate orthodontic treatment or that testosterone deficiency necessarily causes slower tooth movement in humans.
The experimental findings should therefore be interpreted as evidence of a biological relationship that requires further investigation.

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🔹 Important Study Limitation
The principal limitation is the experimental model.
The study was conducted in pubertal male rats, and orthodontic tooth movement was produced experimentally using a closed-coil spring on a maxillary first molar. The observation periods were limited to 5 and 10 days.
Human orthodontic treatment differs substantially from this experimental model in anatomy, skeletal development, treatment duration, biomechanics, hormonal physiology, and clinical management.
Consequently, the findings cannot currently be used to predict how an individual human patient will respond to orthodontic treatment based on testosterone levels.

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💬 Discussion
The 2026 study by Reis et al. provides experimental evidence that testosterone dysfunction can modify bone remodeling, periodontal inflammation, orthodontic tooth movement, and root resorption in a pubertal male rat model.
Both testosterone deficiency and high-dose anabolic-androgenic steroid exposure produced alterations in alveolar bone microarchitecture. High-dose AAS exposure was additionally associated with increased inflammatory infiltration and root resorption, while testosterone disturbances affected several genes and signaling pathways involved in bone metabolism.
These findings expand the experimental understanding of systemic hormonal influences on orthodontic tooth movement. Nevertheless, human clinical studies are required before the findings can be translated into orthodontic treatment recommendations.

📚 References

Reis, C. L. B., Galisteu-Luiz, K., Pedroso, G. L., Puls, G. L., Cassaro, L., Vieira, B. B., Lourenço Romano, F., Küchler, E. C., Kirschneck, C., de Oliveira, D. S. B., Stuani, M. B. S., & Matsumoto, M. A. N. (2026). Effects of testosterone and high-dose anabolic steroids on orthodontic-induced bone remodeling and root resorption: An animal study. Journal of Periodontology. Advance online publication. https://doi.org/10.1002/jper.70068

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Pulp Exposure in Children: Emergency Treatment Guide

Pulp Exposure

Pulp exposure in children is a time-sensitive clinical finding that may occur during deep caries removal, restorative procedures, or dental trauma.

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The appropriate emergency treatment depends primarily on the pulpal diagnosis, extent and cause of exposure, ability to achieve hemostasis, tooth restorability, root development, and presence of infection.

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Current pediatric guidelines emphasize preserving vital pulp tissue whenever predictable healing is possible. However, pulp exposure should not automatically lead to pulpotomy or extraction. A structured diagnosis is essential to select the least invasive treatment with a reasonable prognosis.

Initial Assessment of Pulp Exposure
The emergency assessment should include:
▪️ History: spontaneous pain, provoked pain, duration, nocturnal pain, trauma, and previous symptoms.
▪️ Clinical examination: caries extent, pulp exposure characteristics, swelling, sinus tract, mobility, tenderness, and restorability.
▪️ Radiographic assessment: periapical or bitewing imaging when indicated to evaluate the depth of the lesion, furcation/periapical changes, root resorption, and the developing permanent successor.
▪️ Pulp assessment: determine whether the pulp appears clinically vital and whether bleeding can be controlled after exposure or tissue removal.
In primary teeth, pulpal diagnosis is particularly challenging because conventional sensibility testing has limited reliability. Therefore, clinical and radiographic findings must be interpreted together rather than relying on a single diagnostic test.

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Emergency Treatment Options

1. Direct Pulp Cap
A direct pulp cap (DPC) may be considered when a small pulp exposure occurs in a tooth with a favorable pulpal diagnosis and adequate conditions for maintaining pulp vitality.
The exposed pulp should be protected with a biocompatible pulp-capping material, followed by an effective coronal seal. Calcium-silicate materials are increasingly used because of their biological properties.
However, the indication for DPC differs between primary and permanent teeth. Contemporary evidence supports DPC more strongly in appropriately selected permanent teeth, whereas its routine use for carious exposures in primary teeth is less predictable.

2. Partial Pulpotomy
A partial pulpotomy removes a limited portion of inflamed coronal pulp while preserving the remaining vital tissue.
This approach is particularly valuable for traumatic pulp exposures in immature permanent teeth, where maintaining vitality allows continued root development and apical maturation. IADT guidelines recommend partial pulpotomy for complicated crown fractures with pulp exposure in primary teeth when preservation of the pulp is appropriate, while treatment should be adapted to the child's age, cooperation, and clinical circumstances.

3. Full Pulpotomy
Pulpotomy is one of the principal emergency treatments for a vital primary tooth with pulp exposure when the coronal pulp is inflamed but the radicular pulp remains suitable for treatment.
The contemporary AAPD guideline supports pulpotomy as a vital pulp therapy for primary teeth with appropriate pulpal status. Calcium-silicate-based materials, including mineral trioxide aggregate (MTA), are important contemporary options. The 2024 AAPD guideline specifically recommends against calcium hydroxide as the pulpotomy medicament for primary teeth with deep caries lesions.
A key clinical determinant is hemostasis. Failure to obtain adequate control of pulpal bleeding after appropriate tissue removal should prompt reassessment of the diagnosis and treatment plan rather than simply proceeding with definitive coverage.

4. Pulpectomy or Root Canal Treatment
When the pulp is necrotic or irreversibly diseased, vital pulp therapy is no longer the appropriate treatment.
For a restorable primary tooth, pulpectomy may be indicated when there are clinical or radiographic findings consistent with irreversible pulpitis or necrosis. For permanent teeth, conventional root canal treatment may be required depending on root development and the biological status of the pulp.
In immature permanent teeth, preservation of vitality should remain a priority whenever feasible because it supports continued root maturation and dentinal wall development.

5. Extraction
Extraction should be considered when the tooth is non-restorable, has an unfavorable prognosis, presents extensive pathological root resorption, or cannot be predictably treated because of its clinical condition.
In primary teeth, treatment planning must also consider the stage of exfoliation, space requirements, occlusion, and the developing permanent successor.

Emergency Decision-Making
Clinical Situation Preferred Approach
Small exposure, favorable vital pulp, permanent tooth Direct pulp cap or partial pulpotomy
Traumatic exposure in immature permanent tooth Partial/full pulpotomy to preserve vitality
Vital primary tooth with suitable coronal pulp Pulpotomy
Primary tooth with necrotic or irreversibly diseased pulp Pulpectomy
Non-restorable tooth or unfavorable prognosis Extraction
Uncertain diagnosis Stabilize, diagnose carefully, and refer when necessary
For traumatic exposure of primary teeth, IADT guidance supports partial pulpotomy for complicated crown fractures, while larger exposures may require cervical pulpotomy. In selected cases, definitive treatment can be delayed briefly when rapid referral to a pediatric dental provider is possible.

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Material Selection and Hemostasis
Modern vital pulp therapy increasingly favors calcium-silicate-based biomaterials because of their biocompatibility and bioactive properties. MTA and other hydraulic calcium-silicate cements have demonstrated favorable clinical outcomes, particularly in permanent teeth.
Hemostasis is not merely a technical step; it is an important diagnostic indicator. After appropriate pulp tissue removal, persistent uncontrolled bleeding may indicate more extensive inflammation and should influence treatment selection.
A well-sealed definitive restoration is equally important. Even an appropriately performed pulp therapy can fail if the restoration permits microleakage or bacterial recontamination.

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💬 Discussion
The management of pulp exposure in children has shifted toward biologically based treatment rather than automatically removing all exposed pulp tissue. The current evidence supports a conservative approach when the remaining pulp has the capacity to heal.
For primary teeth, however, clinicians should avoid directly extrapolating evidence from permanent teeth. The 2024 AAPD guideline emphasizes that treatment selection should integrate the pulpal diagnosis, clinical findings, radiographic findings, tooth restorability, and the child's circumstances.
In permanent teeth, particularly immature teeth, preservation of pulp vitality has an additional biological objective: continued root development. Contemporary AAPD recommendations indicate that direct pulp capping, partial pulpotomy, or full pulpotomy using calcium-silicate cement may be considered for selected exposed permanent teeth, including teeth at different stages of root maturation.
Therefore, the emergency objective should not simply be to "cover the nerve." The clinician should determine which pulp tissue remains biologically salvageable and which treatment provides the best opportunity for long-term tooth survival.

🎯 Clinical Recommendations
1. Do not treat pulp exposure as a diagnosis. Establish the pulpal and periapical diagnosis before selecting therapy.
2. Prioritize vital pulp preservation in appropriately selected primary and immature permanent teeth.
3. After pulp exposure, evaluate bleeding control carefully; persistent bleeding should trigger reassessment of the treatment plan.
4. Use calcium-silicate-based materials when indicated for contemporary vital pulp therapy.
5. Ensure an immediate, well-sealed definitive restoration whenever clinically appropriate.
6. In primary teeth, always consider the permanent successor, exfoliation timing, restorability, and space implications.
7. For traumatic exposures, follow established IADT trauma protocols and arrange appropriate follow-up.
8. When diagnosis, cooperation, restorability, or prognosis is uncertain, early referral to a pediatric dentist or endodontist may provide the safest approach.

✍️ Conclusion
Pulp exposure in children requires rapid but biologically guided decision-making. Direct pulp capping, partial pulpotomy, full pulpotomy, pulpectomy, and extraction each have specific indications. Current evidence increasingly supports vital pulp therapy and minimally invasive treatment when the remaining pulp has healing potential. Accurate diagnosis, effective hemostasis, appropriate biomaterials, and a durable coronal seal remain the principal determinants of successful emergency management.

📚 References

✔ American Academy of Pediatric Dentistry. (2024). Use of vital pulp therapies in primary teeth 2024. Pediatric Dentistry, 46(1), 13–26.
✔ American Academy of Pediatric Dentistry. (2025). Guideline for vital pulp therapy in permanent teeth. Pediatric Dentistry, 47(5), 299–311.
✔ American Academy of Pediatric Dentistry. (2026). Pulp therapy for primary and immature permanent teeth: Indications and objectives. In The Reference Manual of Pediatric Dentistry 2026–2027. American Academy of Pediatric Dentistry.
✔ Day, P. F., Flores, M. T., O'Connell, A. C., Abbott, P. V., Tsilingaridis, G., Fouad, A. F., Cohenca, N., Lauridsen, E., Bourguignon, C., Hicks, L., Andreasen, J. O., Cehreli, Z. C., Harlamb, S., Kahler, B., Oginni, A., Semper, M., & Levin, L. (2020). International Association of Dental Traumatology guidelines for the management of traumatic dental injuries: 3. Injuries in the primary dentition. Dental Traumatology, 36(4), 343–359. https://doi.org/10.1111/edt.12576
✔ Duncan, H. F., Galler, K. M., Tomson, P. L., Simon, S., El-Karim, I., Kundzina, R., Krastl, G., Dammaschke, T., Fransson, H., Markvart, M., Zehnder, M., & Bjørndal, L. (2019). European Society of Endodontology position statement: Management of deep caries and the exposed pulp. International Endodontic Journal, 52(7), 923–934. https://doi.org/10.1111/iej.13080

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viernes, 7 de agosto de 2026

Vertical Malocclusions in Primary, Mixed, and Permanent Dentition

Vertical Malocclusions

Vertical malocclusions are bite problems that affect the vertical relationship between the upper and lower teeth. Instead of meeting normally when the mouth closes, the teeth may overlap too much or fail to touch at all.

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These conditions can influence chewing, speech, facial appearance, and jaw function.

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Vertical discrepancies may appear during primary, mixed, or permanent dentition and can result from genetic factors, oral habits, or altered facial growth. Early diagnosis helps guide treatment at the most appropriate stage of development, often reducing the need for more complex procedures later in life.

🔹 What Is a Vertical Malocclusion?
A vertical malocclusion is an abnormal relationship of the teeth or jaws in the vertical dimension. The two most common forms are:

▪️ Deep Bite (Excessive Overbite)
▪️ Anterior Open Bite
These conditions may have a dental origin, a skeletal origin, or a combination of both.

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🔹 Main Types of Vertical Malocclusions

1. Deep Bite (Deep Overbite)
A deep bite occurs when the upper front teeth excessively overlap the lower front teeth, sometimes covering most or all of the lower incisors.
Common characteristics
▪️ Excessive vertical overlap of anterior teeth
▪️ Increased risk of palatal trauma
▪️ Tooth wear
▪️ Functional limitations in severe cases
Deep bite is frequently associated with short lower facial height and strong bite muscles.

2. Anterior Open Bite
An anterior open bite is present when the upper and lower front teeth do not touch when the back teeth are in contact.
Common characteristics
▪️ Space between upper and lower incisors
▪️ Difficulty biting food
▪️ Speech alterations
▪️ Tongue thrusting in some patients
Open bite may be dental, skeletal, or functional.

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🔹 Vertical Malocclusions by Dentition Stage

Primary Dentition
Vertical problems during primary dentition are commonly associated with:
▪️ Thumb sucking
▪️ Prolonged pacifier use
▪️ Tongue thrusting
▪️ Mouth breathing
Many mild cases improve after harmful habits stop, but persistent open bites require professional evaluation.

Mixed Dentition
The mixed dentition provides an excellent opportunity for interceptive orthodontic treatment.
Management may include:
▪️ Habit interception
▪️ Myofunctional therapy
▪️ Orthodontic appliances
▪️ Growth modification when indicated
Early treatment improves the chances of correcting developing skeletal discrepancies.

Permanent Dentition
Once facial growth is complete, treatment becomes more individualized.
Options may include:
▪️ Fixed orthodontic appliances
▪️ Temporary Anchorage Devices (TADs)
▪️ Orthognathic surgery for severe skeletal open bite or deep bite
▪️ Multidisciplinary treatment when necessary

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🔹 Main Causes
Vertical malocclusions often result from multiple factors, including:

▪️ Genetic facial growth patterns
▪️ Thumb or finger sucking
▪️ Prolonged pacifier use
▪️ Tongue thrust habit
▪️ Mouth breathing
▪️ Abnormal eruption patterns
▪️ Neuromuscular factors
▪️ Skeletal growth discrepancies

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🔹 Clinical Signs
Patients may present with:

▪️ Deep Bite
▪️ Excessive incisor overlap
▪️ Lower incisor wear
▪️ Palatal trauma
▪️ Reduced lower facial height

Anterior Open Bite
▪️ Visible gap between front teeth
▪️ Difficulty biting foods
▪️ Speech difficulties
▪️ Tongue thrust during swallowing
▪️ Increased lower facial height in skeletal cases

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🔹 Diagnosis
A comprehensive diagnosis includes:

▪️ Medical and dental history
▪️ Clinical examination
▪️ Facial analysis
▪️ Occlusal evaluation
▪️ Measurement of overbite
▪️ Functional assessment
▪️ Digital models or study casts
▪️ Cephalometric analysis
▪️ CBCT, only when clinically justified
Differentiating dental from skeletal vertical discrepancies is essential for selecting the correct treatment.

🔹 Treatment Overview
Condition Preferred Management
Dental Deep Bite Orthodontic intrusion of anterior teeth, bite-opening mechanics, bite turbos, or arch leveling depending on the patient's needs.
Skeletal Deep Bite Growth modification in growing patients or orthognathic surgery combined with orthodontic treatment in severe adult cases.
Dental Anterior Open Bite Elimination of oral habits, myofunctional therapy when indicated, and orthodontic correction with fixed or removable appliances.
Skeletal Anterior Open Bite Treatment with Temporary Anchorage Devices (TADs), skeletal anchorage mechanics, or orthognathic surgery for severe skeletal discrepancies.
🔹 Possible Consequences if Untreated
Untreated vertical malocclusions may lead to:

▪️ Abnormal tooth wear
▪️ Chewing difficulties
▪️ Speech problems
▪️ Periodontal trauma
▪️ Poor smile aesthetics
▪️ Reduced occlusal stability
▪️ Temporomandibular disorders in susceptible individuals

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💬 Discussion
Recent orthodontic evidence emphasizes that vertical malocclusions require individualized diagnosis, as the underlying cause determines the most effective treatment. While habit-related open bites often respond well to early intervention, skeletal discrepancies may require orthopedic treatment during growth or surgical correction in adulthood. Careful evaluation of facial growth, dental eruption, and oral function is essential for achieving stable long-term results.

🎯 Recommendations
▪️ Perform routine orthodontic screening during childhood.
▪️ Eliminate thumb sucking and prolonged pacifier use early.
▪️ Monitor tongue posture and swallowing patterns.
▪️ Refer patients with persistent open bite or deep bite for orthodontic evaluation.
▪️ Base treatment decisions on growth stage and skeletal diagnosis rather than age alone.
▪️ Use advanced imaging only when clinically indicated.

✍️ Conclusion
Vertical malocclusions are common developmental conditions that may affect patients from primary through permanent dentition. Their impact extends beyond tooth alignment, influencing facial growth, oral function, and long-term stability. Early identification of harmful habits and accurate differentiation between dental and skeletal causes allow clinicians to provide more effective, less invasive treatment and improve overall oral health outcomes.

📊 Summary Table
Aspect Key Information
Definition Abnormal vertical relationship between the upper and lower teeth or jaws.
Main Types Deep bite (deep overbite) and anterior open bite.
Primary Dentition Often associated with thumb sucking, prolonged pacifier use, tongue thrusting, and mouth breathing.
Mixed Dentition Ideal stage for interceptive orthodontics, habit control, and growth modification.
Permanent Dentition Treatment may include fixed appliances, TADs, or orthognathic surgery depending on severity.
Main Causes Genetics, oral habits, tongue thrust, mouth breathing, and skeletal growth discrepancies.
Diagnosis Clinical examination, facial analysis, overbite measurement, study models, cephalometric analysis, and CBCT when indicated.
Early Intervention Improves treatment outcomes, supports normal facial growth, and may reduce the need for surgery later.

📚 References

✔ Graber, L. W., Vanarsdall, R. L., Vig, K. W. L., & Huang, G. J. (2023). Orthodontics: Current Principles and Techniques (7th ed.). Elsevier.
✔ Proffit, W. R., Fields, H. W., Larson, B. E., & Sarver, D. M. (2023). Contemporary Orthodontics (7th ed.). Elsevier.
✔ Ngan, P., & Fields, H. W. (1997). Open bite: A review of etiology and management. Pediatric Dentistry, 19(2), 91–98.
✔ Greenlee, G. M., Huang, G. J., Chen, S. S., Chen, J., Koepsell, T., & Hujoel, P. (2011). Stability of treatment for anterior open-bite malocclusion: A meta-analysis. American Journal of Orthodontics and Dentofacial Orthopedics, 139(2), 154–169.
✔ American Association of Orthodontists. (2024). Clinical Practice Resources. American Association of Orthodontists.

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