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jueves, 11 de diciembre de 2025

Understanding Dental Erosion: Causes and Prevention (Updated Guide)

Dental Erosion

Dental erosion is a progressive, non-carious loss of dental hard tissue caused by acidic dissolution without bacterial involvement. Unlike dental caries, dental erosion results from chemical wear driven by dietary acids, intrinsic acids, and lifestyle factors.

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Understanding the causes and prevention of dental erosion is essential for early diagnosis, long-term oral health, and effective patient education.

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1. What Is Dental Erosion?
Dental erosion refers to the irreversible loss of enamel and dentin caused by exposure to non-bacterial acids. Over time, this leads to thinning enamel, dentin hypersensitivity, aesthetic changes, and increased susceptibility to mechanical wear. Early identification is crucial to minimize long-term consequences.

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2. Causes of Dental Erosion

2.1 Dietary Acids
Acidic beverages and foods—such as soft drinks, citrus fruits, sports drinks, and vinegar—are significant risk factors. Frequent consumption of acidic beverages dramatically increases erosion progression.

2.2 Intrinsic Acids
Conditions like gastroesophageal reflux disease (GERD), chronic vomiting, and eating disorders expose teeth to gastric acids with a pH of 1–2, causing severe erosive wear, especially on palatal surfaces.

2.3 Environmental and Behavioral Factors
▪️ Occupational exposure (e.g., wine tasting, battery factory workers).
▪️ Dry mouth and reduced buffering capacity.
▪️ Aggressive brushing immediately after acid exposure.

These factors contribute to accelerated enamel breakdown.

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3. Prevention of Dental Erosion

3.1 Dietary Modifications
Reducing acidic beverage intake and avoiding sipping throughout the day significantly lowers erosion risk. Encourage patients to consume acidic drinks with meals and use a straw.

3.2 Enhancing Salivary Protection
Adequate hydration, sugar-free gum, and managing xerostomia enhance natural buffering capacity.

3.3 Fluoride and Remineralization Therapy
Topical fluorides, CPP-ACP, and high-fluoride varnishes increase enamel resistance to acid attack. Professional application plays a crucial preventive role.

3.4 Behavioral and Oral Hygiene Recommendations
Patients should avoid brushing for at least 30 minutes after acid exposure, use soft-bristled brushes, and choose non-abrasive toothpaste.

📊 Comparative Table: Key Clinical Factors in Dental Erosion Prevention

Aspect Advantages Limitations
Fluoride Therapy Enhances enamel resistance; reduces erosion progression Requires repeated applications; limited effect without behavior change
Dietary Modifications Reduces acid exposure; improves long-term outcomes Patient compliance varies; lifestyle habits hard to change
Salivary Stimulation Improves natural buffering; protects against acid attacks Less effective in patients with xerostomia or systemic conditions
Behavioral Adjustments Simple, low-cost strategies; reduces mechanical wear Effectiveness depends heavily on patient adherence
💬 Discussion

Dental erosion remains a growing global concern due to lifestyle changes, increased consumption of acidic beverages, and rising rates of GERD in both adults and children. The interplay between dietary habits, intrinsic acids, and behavioral factors requires a multifactorial approach. Prevention must focus on patient education, regular monitoring, and targeted remineralization therapies.
Clinicians should recognize erosion patterns early and differentiate them from abrasion and attrition. Without timely intervention, erosion can lead to dentin hypersensitivity, aesthetic deterioration, and costly restorative treatments.

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✍️ Conclusion
Dental erosion is a preventable yet progressive condition. Identifying its causes—dietary, intrinsic, and environmental—is essential for effective clinical management. Preventive strategies, patient education, and early professional intervention significantly reduce long-term damage. A combination of lifestyle modifications, saliva promotion, and remineralization therapies provides the strongest defense against erosive tooth wear.

🔎 Recommendations
▪️ Limit acidic beverages and avoid prolonged sipping.
▪️ Implement remineralization therapies such as fluoride varnishes and CPP-ACP.
▪️ Encourage salivary stimulation and hydration.
▪️ Avoid aggressive brushing immediately after consuming acids.
▪️ Monitor high-risk patients (GERD, xerostomia, eating disorders) more frequently.

📚 References

✔ Bartlett, D. W., Lussi, A., & West, N. X. (2013). Dental erosion: An overview of its etiology, diagnosis, and management. Dental Clinics of North America, 57(3), 495–512. https://doi.org/10.1016/j.cden.2013.04.006
✔ Lussi, A., & Carvalho, T. S. (2014). Erosive tooth wear: A multifactorial condition of growing concern and increasing knowledge. Monographs in Oral Science, 25, 1–15. https://doi.org/10.1159/000360380
✔ West, N. X., Joiner, A., & Brookes, S. J. (2018). Enamel erosion: Prevention and therapy. Clinical Oral Investigations, 22(2), 821–831. https://doi.org/10.1007/s00784-017-2321-7

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Fluoride Safety in Children: What Parents Need to Know (Updated Guide)

Fluoride

Fluoride remains one of the most effective, safe, and widely recommended measures to prevent dental caries in children. Despite its proven benefits, many parents still express concerns about dose, toxicity, and appropriate use.

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Updated guidelines from the American Dental Association (ADA) and the American Academy of Pediatric Dentistry (AAPD) emphasize that fluoride is safe when used correctly, making parental education essential.

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Why Fluoride Matters for Children
Dental caries is the most common chronic disease in childhood, and fluoride significantly reduces its incidence. Topical fluoride strengthens enamel, enhances remineralization, and inhibits bacterial activity in dental plaque.

➤ Key Benefits
▪️ Reduces caries risk by 25–30%
▪️ Safe when applied in recommended concentrations
▪️ Works on both erupted and developing teeth
▪️ Supports long-term oral health and reduces treatment needs

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Understanding Fluoride Safety

1. Recommended Daily Exposure
The ADA and AAPD recommend 0.05 mg/kg/day as a safe intake threshold for children. Excessive ingestion may lead to dental fluorosis, but only during the early developmental years.

2. Safe Use of Fluoride Toothpaste
▪️ Children less than 3 years: a “smear” or rice-sized amount (1000 ppm)
▪️ Children 3–6 years: pea-sized amount (1000–1450 ppm)
▪️ Supervision is essential to prevent swallowing.

3. Professional Fluoride Applications
In-office treatments use higher concentrations but are clinically safe due to controlled application. These include:
▪️ 5% sodium fluoride varnish (22,600 ppm)
▪️ 1.23% acidulated phosphate fluoride (APF) gel
▪️ 2% neutral sodium fluoride (NaF) gel

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Common Parental Myths and Evidence-Based Responses

➤ Myth 1: “Fluoride is toxic for children.”
Reality: True toxicity occurs only at extremely high doses. Therapeutic use is far below toxic thresholds.

➤ Myth 2: “Fluoride causes harm even in toothpaste form.”
Reality: Fluoride toothpaste is safe when used in age-appropriate amounts.

➤ Myth 3: “Varnish applications are dangerous.”
Reality: Varnishes release fluoride gradually, minimizing systemic exposure.

📊 Comparative Table: Fluoride Presentations in the Dental Office

Aspect Advantages Limitations
5% Sodium Fluoride Varnish (22,600 ppm) Highly effective; minimal ingestion risk; ideal for young children Temporary yellow coloration; requires professional application
1.23% APF Gel Strong enamel uptake; fast application Not recommended for children under 6 due to swallowing risk; etches ceramic restorations
2% Neutral Sodium Fluoride Gel Safe for patients with restorations; good uptake Requires trays; higher ingestion risk in younger children
Fluoride Foam Less material used; reduces ingestion Lower fluoride contact time compared to gel
Silver Diamine Fluoride (38%) Arrests caries effectively; non-invasive Causes black staining; requires informed consent
💬 Discussion
Current evidence strongly supports the safety and effectiveness of fluoride in preventing childhood caries. Concerns typically arise from misuse—particularly swallowing large quantities of toothpaste or supplement overuse. Professional application remains a gold standard for high-risk patients, and patient education plays a critical role in ensuring safe home use.

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🎯 Recommendations for Parents

▪️ Use fluoride toothpaste twice daily, following age-based dosing.
▪️ Schedule professional fluoride applications every 3–6 months for high-risk children.
▪️ Avoid fluoride mouthrinses for children under 6 years.
▪️ Store dental products out of reach to prevent accidental ingestion.
▪️ Choose fluoridated community water when available.

✍️ Conclusion
Fluoride is a safe, effective, and essential tool in preventing dental caries in children. When used properly in both home and clinical settings, it poses minimal risk while offering significant protective benefits. Updated evidence continues to confirm that fluoride remains one of the most important public health measures for children’s oral health.

📚 References

✔ American Academy of Pediatric Dentistry. (2023). Fluoride therapy guideline. AAPD. https://www.aapd.org
✔ American Dental Association. (2021). Fluoride: Topical and systemic supplements. ADA Council on Scientific Affairs. https://www.ada.org
✔ Weyant, R. J., Tracy, S. L., Anselmo, T. T., Beltrán-Aguilar, E., Donly, K. J., & Frese, W. A. (2013). Topical fluoride for caries prevention. Journal of the American Dental Association, 144(11), 1279–1291. https://doi.org/10.14219/jada.archive.2013.0057
✔ Meyer-Lueckel, H., Paris, S., & Ekstrand, K. (2013). Caries management – science and clinical practice. Thieme.

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miércoles, 10 de diciembre de 2025

Amoxicillin in Pediatric Dentistry: Evidence-Based Uses and Safety Recommendations

Amoxicillin

Amoxicillin remains the first-line antibiotic in pediatric dentistry due to its broad-spectrum activity, excellent safety profile, and effectiveness against common odontogenic pathogens.

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However, modern guidelines emphasize that antibiotics should not replace dental treatment and must be prescribed only when truly indicated. Understanding evidence-based uses, dosing recommendations, and limitations is essential to avoid overprescription and antimicrobial resistance.

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When Is Amoxicillin Indicated in Pediatric Dentistry?
According to the American Academy of Pediatric Dentistry (AAPD) and the American Dental Association (ADA), amoxicillin is indicated only in infections with systemic involvement, facial swelling, or when the infection spreads beyond the local tooth structure.

Key Indications
▪️ Acute odontogenic infections with systemic signs (fever, malaise)
▪️ Facial cellulitis
▪️ Acute apical abscess with systemic involvement
▪️ Lymphadenitis associated with dental infection
▪️ Postoperative management in select surgical cases (not routine)

Amoxicillin is not indicated for:
▪️ Localized pulpitis
▪️ Reversible or irreversible pulpitis without swelling
▪️ Routine pulpotomies or pulpectomies
▪️ Dental pain without infection

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Mechanism of Action
Amoxicillin is a β-lactam antibiotic that inhibits bacterial cell wall synthesis. It has strong activity against:

▪️ Streptococcus mutans
▪️ Streptococcus anginosus group
▪️ Prevotella species (partially)
It is less effective against β-lactamase–producing organisms unless combined with clavulanic acid.

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Pediatric Dosage Recommendations
AAPD guidelines recommend:

▪️ Children (less than 40 kg):
20–40 mg/kg/day divided every 8 hours or 25–45 mg/kg/day divided every 12 hours
▪️ Children ≥40 kg:
Standard adult dosing (500 mg every 8 hours)

Treatment duration typically lasts 5–7 days, with emphasis on clinical improvement within 48–72 hours.

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Safety Profile and Contraindications
Amoxicillin is considered safe, but clinicians must be aware of:

➤ Adverse Reactions
▪️ Mild gastrointestinal symptoms
▪️ Rash
▪️ Hypersensitivity reactions
▪️ Rare anaphylaxis in penicillin-allergic patients

➤ Contraindications
▪️ Confirmed penicillin allergy
▪️ Previous severe reaction to β-lactams

📊 Comparative Table: Amoxicillin Efficacy in Odontogenic Infections

Aspect Advantages Limitations
Acute Apical Abscess with Systemic Involvement High efficacy against Streptococcus species; reduces systemic symptoms Does not eliminate infection without drainage or pulpal treatment
Facial Cellulitis Strong action against predominant pathogens; rapid improvement in swelling Limited effect against β-lactamase–producing anaerobes
Lymphadenitis of Odontogenic Origin Effective in reducing lymph node tenderness and inflammation Requires elimination of dental source to prevent recurrence
Periodontal Abscess (Pediatric) Adjunctive use reduces systemic manifestations Not effective without mechanical debridement
Pericoronitis (Mixed Dentition) Useful when systemic signs present Resistance observed in anaerobic flora; requires irrigation
Odontogenic Sinus Tract with Infection Spread Helps control systemic spread; improves healing after root canal/extraction Ineffective alone; definitive treatment required
💬 Discussion
Although amoxicillin is highly effective for pediatric odontogenic infections, studies show that nearly 80% of dental antibiotic prescriptions are unnecessary. Overuse contributes to resistant strains such as β-lactamase–producing Prevotella.
Correct diagnosis, radiographic evaluation, and definitive dental treatment remain the cornerstone of care. Antibiotics serve as an adjunct, not a substitute, for pulp therapy, drainage, or extraction.

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🔎 Recommendations
1. Prescribe amoxicillin only when systemic involvement is present.
2. Always perform definitive treatment (pulpotomy, pulpectomy, extraction) regardless of antibiotic use.
3. Reassess within 48–72 hours to confirm improvement.
4. For patients with penicillin allergy, consider clindamycin or azithromycin.
5. Avoid prescribing antibiotics for dental pain without infection.

✍️ Conclusion
Amoxicillin remains a highly effective and safe option for managing pediatric odontogenic infections, provided it is used responsibly and following current evidence-based guidelines. Proper diagnosis and definitive dental treatment, alongside prudent antibiotic use, ensure optimal outcomes and minimize antimicrobial resistance.

📚 References

✔ American Academy of Pediatric Dentistry. (2023). Use of antibiotic therapy for pediatric dental patients. AAPD Clinical Guidelines. https://www.aapd.org
✔ Dar-Odeh, N., Fadel, H., Abu-Hammad, S., Abdeljawad, R., & Abu-Hammad, O. (2018). Antibiotic prescribing for dental infections: A review. British Dental Journal, 225(5), 353–359. https://doi.org/10.1038/sj.bdj.2018.733
✔ Robertson, D., & Smith, A. J. (2009). The microbiology of the acute dental abscess. Journal of Medical Microbiology, 58(2), 155–162. https://doi.org/10.1099/jmm.0.003517-0
✔ Segura-Egea, J. J., Gould, K., & Şen, B. H. (2017). Antibiotics in endodontics: A review. International Endodontic Journal, 50(12), 1169–1184. https://doi.org/10.1111/iej.12868

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Pulp Necrosis in Primary Teeth: Diagnosis and Evidence-Based Management

Pulp Necrosis

Pulp necrosis in primary teeth is a common consequence of untreated caries, trauma, or chronic inflammation.

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Early detection is crucial to prevent infection, pathological root resorption, and damage to the developing permanent successor. Understanding accurate diagnostic criteria and selecting the appropriate evidence-based treatment is essential for successful outcomes in pediatric patients.

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Etiology of Pulp Necrosis in Primary Teeth
Primary teeth are particularly vulnerable to necrosis due to:

▪️ Extensive caries progression
▪️ Anatomically thinner enamel and dentin
▪️ Traumatic dental injuries
▪️ Bacterial invasion of the pulp chamber
▪️ Chronic pulpal inflammation progressing to necrosis

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Clinical and Radiographic Diagnosis
Accurate diagnosis relies on a combination of:

➤ Patient history (spontaneous pain, swelling, trauma)

➤ Clinical signs
▪️ Tooth discoloration (grayish)
▪️ Sinus tract
▪️ Tenderness to percussion
▪️ Gingival swelling

➤ Radiographic findings
▪️ Furcation radiolucency
▪️ Pathological root resorption
▪️ Periodontal space widening
▪️ Loss of lamina dura

In primary teeth, pulp testing methods like thermal or electric tests are unreliable, making radiographic and clinical findings essential.

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Management of Pulp Necrosis in Primary Teeth
Once necrosis is confirmed, vital pulp therapies are contraindicated. Treatment options include:

➤ Pulpectomy
The preferred treatment when the tooth is restorable and the infection can be adequately controlled.
Key principles:
▪️ Thorough canal debridement
▪️ Irrigation with 2.5% sodium hypochlorite or chlorhexidine
▪️ Obturating canals with resorbable materials such as iodoform-based pastes (Vitapex/Metapex)
▪️ Final restoration, ideally with a stainless steel crown

➤ Extraction
Indicated when:
▪️ The tooth is non-restorable
▪️ There is excessive root resorption
▪️ Infection compromises the permanent successor
▪️ Patient cooperation is insufficient

➤ Antibiotics
Not routinely recommended unless:
▪️ Facial cellulitis
▪️ Systemic involvement (fever, malaise)
▪️ Spread of infection

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Prognosis
Successful pulpectomy has a 70–85% success rate, depending on canal anatomy, irrigation protocol, and obturation material. Failure occurs when infection persists, when restoration is inadequate, or when premature root resorption affects sealing.

📊 Comparative Table: Pulpectomy vs Extraction in Necrotic Primary Teeth

Aspect Advantages Limitations
Pulpectomy Preserves arch space; maintains function; prevents premature tooth loss; avoids malocclusions Technique sensitive; lower success in multirooted teeth; requires patient cooperation
Extraction Quick relief of infection; avoids complex instrumentation; suitable for non-restorable teeth Loss of arch space; risk of mesial drift; possible need for space maintainers
💬 Discussion
Managing necrotic primary teeth requires careful consideration of the child's age, behavior, the strategic value of the tooth, and the proximity to the permanent successor. Resorbable obturation materials remain essential, as non-resorbable materials can interfere with exfoliation. Current research supports the use of iodoform-based pastes due to superior antibacterial activity and predictable resorption.

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🔎 Recommendations
▪️ Use radiographic criteria as the primary diagnostic tool for suspected necrosis.
▪️ Select pulpectomy when infection is localized and the tooth is strategically important.
▪️ Extract teeth with advanced pathology or poor restorative prognosis.
▪️ Avoid antibiotics unless systemic involvement is present.
▪️ Always protect the treated tooth with a stainless steel crown to ensure long-term success.

✍️ Conclusion
Pulp necrosis in primary teeth requires timely diagnosis and evidence-based intervention to prevent complications and safeguard the developing dentition. Pulpectomy remains the treatment of choice for restorable necrotic teeth, while extraction is indicated in severe cases. Proper case selection and modern pediatric endodontic protocols significantly improve outcomes.

📚 References

✔ American Academy of Pediatric Dentistry. (2021). Pulp therapy for primary and immature permanent teeth. AAPD Clinical Guidelines. https://www.aapd.org
✔ Barja-Fidalgo, F., Ribeiro, A., & Pauleto, A. (2011). Clinical and radiographic outcome of primary molars pulpectomies using different filling materials. Journal of Clinical Pediatric Dentistry, 35(4), 359–364. https://doi.org/10.17796/jcpd.35.4.j1471781m0011654
✔ Fuks, A. B. (2008). Pulp therapy for the primary dentition. Pediatric Dentistry, 30(3), 230–236.
✔ Nadin, G., Goel, B. R., & Yeung, C. A. (2003). Pulp treatment for primary teeth. Cochrane Database of Systematic Reviews, (1), CD003220. https://doi.org/10.1002/14651858.CD003220

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Pulp Therapy

Pulp therapy in primary teeth is a cornerstone of pediatric dentistry, aiming to maintain tooth vitality, prevent infection, and preserve arch space until natural exfoliation.

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Advances in biomaterials such as MTA, Biodentine, and improved clinical protocols have significantly increased success rates. Understanding the differences between pulpotomy, pulpectomy, indirect pulp treatment (IPT), and apexification is essential for evidence-based care.

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Pulp Therapy Techniques

➤ Indirect Pulp Treatment (IPT)
IPT is indicated when deep caries is present but the tooth remains vital and asymptomatic. Selective caries removal minimizes pulp exposure and promotes remineralization. High-fluoride glass ionomer and resin-modified glass ionomer (RMGI) are widely used as liners.

➤ Direct Pulp Cap (DPC)
Used when a small mechanical pulp exposure occurs. Bioceramics like MTA and Biodentine create a durable dentin bridge and exhibit excellent biocompatibility.

➤ Pulpotomy
Indicated in cases of carious pulp exposure with preserved radicular pulp vitality. Popular medicaments include MTA, Biodentine, and historically formocresol, although the latter is no longer recommended due to toxicity concerns.

➤ Pulpectomy
Indicated for irreversible pulpitis or necrosis. It consists of removing necrotic tissue and obturating canals with resorbable materials such as iodoform-based pastes (Vitapex, Metapex) or zinc oxide–eugenol.

➤ Apexogenesis & Apexification in Young Permanent Teeth
Although not used in primary teeth, they are fundamental when treating immature permanent teeth with open apices.

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Materials Used in Pulp Therapy

➤ Mineral Trioxide Aggregate (MTA)
Known for high biocompatibility, antibacterial properties, and superior long-term sealing.

➤ Biodentine
A bioactive dentin substitute with faster setting time and strong pulpal healing potential.

➤ Zinc Oxide–Eugenol (ZOE)
Traditional obturation material for primary teeth, but less favorable in cases requiring complete resorption.

➤ Iodoform-based Pastes (Vitapex/Metapex)
Preferred for pulpectomy due to their resorbability and antimicrobial action.

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Success Rates

▪️ IPT: 90–97% (AAPD, 2021)
▪️ Pulpotomy with MTA: 90–95%
▪️ Pulpotomy with Biodentine: 88–94%
▪️ Pulpectomy: 70–85%, depending on canal anatomy and material used

📊 Comparative Table: Differences Between Pulp Therapy Techniques

Aspect Advantages Limitations
Indirect Pulp Treatment (IPT) High success rates; preserves vitality; minimally invasive Requires excellent diagnosis; risk of residual caries
Direct Pulp Cap (DPC) Promotes dentin bridge formation; effective with bioceramics Not suitable for carious exposures; requires ideal isolation
Pulpotomy High success with MTA/Biodentine; preserves radicular pulp vitality Failure if radicular pulp is inflamed or infected
Pulpectomy Indicated for necrotic teeth; removes infection; allows tooth preservation Technique sensitive; lower success rates; requires resorbable obturants
Apexogenesis Allows continued root development Not applicable to primary teeth
Apexification Induces apical closure in young permanent teeth Long treatment time; not used in primary teeth
💬 Discussion
The choice of pulp therapy depends on diagnosis, degree of inflammation, tooth restorability, and patient behavior. Vital pulp therapies (IPT, DPC, pulpotomy) consistently show higher long-term success than pulpectomy. Modern biomaterials like MTA and Biodentine have replaced older agents due to improved healing outcomes and safety profiles.

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🔎 Recommendations
▪️ Prioritize vital pulp therapies when pulp vitality is preserved.
▪️ Use bioceramics (MTA, Biodentine) as first-line agents.
▪️ Perform pulpectomy only when irreversible pulpitis or necrosis is confirmed.
▪️ Seal treated teeth with stainless steel crowns for long-term success.
▪️ Follow AAPD guidelines for diagnostic criteria and material selection.

✍️ Conclusion
Pulp therapy in primary teeth is highly successful when clinicians use accurate diagnostic criteria and evidence-based materials. Modern biomaterials have improved outcomes and reduced complications, making pulp conservation the preferred approach whenever possible. A clear understanding of each technique ensures predictable and biologically sound results.

📚 References

✔ American Academy of Pediatric Dentistry. (2021). Pulp therapy for primary and immature permanent teeth. AAPD Clinical Guidelines. https://www.aapd.org
✔ Hegde, S., & Bhat, S. S. (2019). Clinical evaluation of MTA and Biodentine as pulpotomy agents in primary teeth. Journal of Indian Society of Pedodontics and Preventive Dentistry, 37(3), 307–315. https://doi.org/10.4103/JISPPD.JISPPD_217_18
✔ Jeon, H. J., Kim, J., & Kim, Y. (2020). Outcomes of vital pulp therapy using bioceramic materials. Restorative Dentistry & Endodontics, 45(3), e32. https://doi.org/10.5395/rde.2020.45.e32
✔ Nowicka, A., Lipski, M., Parafiniuk, M., et al. (2013). Biodentine vs. MTA in direct pulp capping. Journal of Endodontics, 39(6), 743–747. https://doi.org/10.1016/j.joen.2013.01.005

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